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Epidemiological studies have revealed concurrence of specific cancers with other disease states such as metabolic syndrome, inflammatory disease and autoimmune disease. Patients with these chronic conditions have a higher incidence of various cancers, more aggressive tumors, and a higher mortality r...
ORGANISM(S): Homo sapiens 
PolyA+ RNA from retinoic acid stimulated HL-60 cells at 0, 2, 8, or 32 hours using Affymetrix ENCODE tiling chip with NCBI build 35 annotation. PolyA+ RNA from retinoic acid stimulated HL-60 cells at 0, 2, 8, or 32 hours.
ORGANISM(S): Homo sapiens 
p63 with and without actinomycin D treatment ChIP from retinoic acid stimulated ME180 Cells at 0 hours using Affymetrix ENCODE tiling chip with NCBI build 35 annotation. p63_ActD and p63_mActD ChIP from retinoic acid stimulated ME180 Cells, 0 hours.
ORGANISM(S): Homo sapiens 
H3K9K14D ChIP-chip of retinoic acid-stimulated HL60 cells at 0, 2, 8, or 32 hours using Affymetrix ENCODE tiling chip with NCBI build 35 annotation. H3K9K14D ChIP-chip of retinoic acid-stimulated HL60 cells at 0, 2, 8, or 32 hours.
ORGANISM(S): Homo sapiens 
HisH4 ChIP-chip of retinoic acid-stimulated HL60 cells at 0, 2, 8, or 32 hours using Affymetrix ENCODE tiling chip with NCBI build 35 annotation. HisH4 ChIP-chip of retinoic acid-stimulated HL60 cells at 0, 2, 8, or 32 hours.
ORGANISM(S): Homo sapiens 
Pol2 ChIP-chip of retinoic acid-stimulated HL60 cells at 0, 2, 8, or 32 hours using Affymetrix ENCODE tiling chip with NCBI build 35 annotation. Pol2 ChIP-chip of retinoic acid-stimulated HL60 cells at 0, 2, 8, or 32 hours.
ORGANISM(S): Homo sapiens 
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