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TranscriptomicsExpression profiling of laser-microdissected tissue from Tg2576 plaque-depositing mouse model of Alzheimer's disease
Amyloid-M-CM-^_ (AM-CM-^_) plaques are pathological hallmarks of Alzheimer disease. However, the precise neuropathological changes that occur in brain in response to amyloid deposition are largely unknown. To study the molecular mechanism(s) responsible for AM-CM-^_-mediated neuropathology, we perfo...
ORGANISM(S): Mus musculus 
2014-11-04 | E-GEOD-15056 | biostudies-arrayexpress
TranscriptomicsGene expression profiling of in vitro matured bovine oocytes cultivated with or without cumulus cells.
Cumulus cells, surrounding the oocyte, play a key role in the acquisition of oocyte competence to be fertilized and to sustain early embryo development. Cumulus cells contribute to oocyte development by metabolizing energy substrates such as glutathione that may protect the oocyte from oxidative str...
ORGANISM(S): Bos taurus 
2011-08-16 | E-GEOD-31361 | biostudies-arrayexpress
TranscriptomicsExpression data from human carcinoma (MCF7) derived cells that have been exposed to insulin analogues
Insulin analogues are designed to improve the pharmacokinetic parameters compared to regular human insulin. This provides a sustained control of blood glucose levels in diabetic patients. All novel insulin analogues are tested for their mitogenic side effects, however these assays do not take into a...
ORGANISM(S): Homo sapiens 
2015-01-29 | E-GEOD-65398 | biostudies-arrayexpress
TranscriptomicsProfiling of circulating microRNAs in patients with Barrettâ??s esophagus and esophageal adenocarcinoma
miRNAs were extracted from plasma samples of healthy volunteers, patients with Barrettâ??s esophagus and patients with esophageal adenocarcinoma. We used the Serum / Plasma Focus miRNA PCR panel (Exiqon, Denmark) to quantitate miRNA presence in the different patient groups. PCR miRNA profiling. miRN...
ORGANISM(S): Homo sapiens 
2015-11-19 | E-GEOD-51410 | biostudies-arrayexpress
UnknownHeterozygous STAT1 gain-of-function mutations underlie an unexpectedly broad clinical phenotype.
Not available
S-EPMC4920021 | biostudies-literature
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