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Collège de France, Oncologie Cellulaire et Moléculaire, PSL University, INSERM UMR 1050, CNRS UMR 7241, INSERM UMR 944, CNRS UMR 7212, Université de Paris, IRSL, Hôpital St. Louis Paris France
(1)
INSERM U1016, Institut Cochin, CNRS UMR8104, Paris, France; Université Paris Descartes, Faculté de Médecine Sorbonne Paris Cité, Paris, France; Equipe Labellisée Ligue Nationale Contre le Cancer (LNCC), Paris, France.
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ITGBL1 is a new immunomodulator that favors development of melanoma tumors by inhibiting natural killer cells cytotoxicity.
Not available
S-EPMC7789764
|
biostudies-literature
Cite
Conditional Gene Targeting Reveals Cell Type-Specific Roles of the Lysosomal Protease Cathepsin L in Mammary Tumor Progression.
Not available
S-EPMC7463523
|
biostudies-literature
Cite
The small heat shock protein B8 (HSPB8) confers resistance to bortezomib by promoting autophagic removal of misfolded proteins in multiple myeloma cells.
Not available
S-EPMC4171627
|
biostudies-literature
Cite
Case Report: Acquired Disseminated BCG in the Context of a Delayed Immune Reconstitution After Hematological Malignancy.
Not available
S-EPMC8369751
|
biostudies-literature
Cite
Correction to: ITGBL1 is a new immunomodulator that favors development of melanoma tumors by inhibiting natural killer cells cytotoxicity.
Not available
S-EPMC7839296
|
biostudies-literature
Cite
CD271 is an imperfect marker for melanoma initiating cells.
Not available
S-EPMC4170612
|
biostudies-literature
Cite
Mechanisms of AXL overexpression and function in Imatinib-resistant chronic myeloid leukemia cells.
Not available
S-EPMC3259992
|
biostudies-literature
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Individual differences in executive functions and theory of mind mediate the relation between academic skills from kindergarten to 5th grade.
Not available
S-EPMC12129330
|
biostudies-literature
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Improving nutritional status after allogeneic stem cell transplantation: results of phase 2 ALLONUT clinical trial.
Not available
S-EPMC11226397
|
biostudies-literature
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Innate lymphocyte-induced CXCR3B-mediated melanocyte apoptosis is a potential initiator of T-cell autoreactivity in vitiligo.
Not available
S-EPMC6522502
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biostudies-literature
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