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TranscriptomicsTranscription profiling of human HeLa cells treated with major listerial toxin listeriolysin
Upon infection, pathogens reprogram host gene expression. In eukaryotic cells, genetic reprogramming is induced by the concerted activation/repression of transcription factors and various histone modifications that control DNA accessibility in chromatin. We report here that the bacterial pathogen, ...
ORGANISM(S): Homo sapiens 
2008-04-01 | E-MEXP-1078 | biostudies-arrayexpress
TranscriptomicsListeria monocytogenes induces T cell receptor unresponsiveness via its pore-forming toxin listeriolysin O
The success of many pathogens relies on their ability to circumvent the innate and adaptive immune defenses. How bacterial pathogens subvert host responses is not clear. Cholesterol-dependent cytolysins (CDCs) represent an expansive family of homologous pore-forming toxins produced by more than 20 G...
ORGANISM(S): Mus musculus 
2010-07-29 | E-GEOD-19337 | biostudies-arrayexpress
ProteomicsShort-term remodeling of the host epithelial cell proteome by the listeriolysin O pore-forming toxin
Bacterial pathogens use various strategies to interfere with host cell functions. Among these strategies, bacteria induce transcriptional changes, in order to modify the set of proteins synthetized by the host cell, or target pre-existing proteins by modulating their post-translational modifications...
ORGANISM(S): Homo sapiens (Human) 
2018-05-17 | PXD009339 | Pride
Pore-forming toxin Listeriolysin o Bacterial infection Listeria 
ProteomicsBacterial pore-forming toxins remodel the host epithelial cell proteome
Bacterial pathogens use various strategies to interfere with host cell functions. Among these strategies, bacteria induce transcriptional changes, in order to modify the set of proteins synthetized by the host cell, or target pre-existing proteins by modulating their post-translational modifications...
ORGANISM(S): Homo sapiens (Human) 
2018-11-07 | PXD006996 | Pride
Listeria Infection Listeriolysin o 
TranscriptomicsListeria host responses
This logical set encompases several 8hr timecourses (0,1,2,4,8 hrs) and their replicates. All correspond to treatments of bone marrow derived macrophages. Abstract: Innate and adaptive immunity depends critically on host recognition of pathogen-associated molecules. Toll-like receptors (TLRs) are k...
ORGANISM(S): Mus musculus 
2006-01-06 | E-GEOD-3991 | biostudies-arrayexpress
TranscriptomicsListeria monocytogenes induces T cell receptor unresponsiveness via its pore-forming toxin listeriolysin O
The success of many pathogens relies on their ability to circumvent the innate and adaptive immune defenses. How bacterial pathogens subvert host responses is not clear. Cholesterol-dependent cytolysins (CDCs) represent an expansive family of homologous pore-forming toxins produced by more than 20 G...
ORGANISM(S): Mus musculus 
2010-06-29 | GSE19337 | GEO
ProteomicsBacterial pore-forming toxins remodel the host epithelial cell proteome
Bacterial pathogens use various strategies to interfere with host cell functions. Among these strategies, bacteria induce transcriptional changes, in order to modify the set of proteins synthetized by the host cell, or target pre-existing proteins by modulating their post-translational modifications...
ORGANISM(S): Homo sapiens (Human) 
2018-11-07 | PXD006900 | Pride
Listeria infection Pore-forming toxin Silac Listeriolysin o 
UnknownRole in virulence of phospholipases, listeriolysin O and listeriolysin S from epidemic Listeria monocytogenes using the chicken embryo infection model.
Not available
S-EPMC5801685 | biostudies-literature
UnknownCrystallization and X-ray crystallographic analysis of the cholesterol-dependent cytolysin listeriolysin O from Listeria monocytogenes.
Not available
S-EPMC3818035 | biostudies-literature
UnknownListeriolysin O-dependent host surfaceome remodeling modulates Listeria monocytogenes invasion.
Not available
S-EPMC6282100 | biostudies-literature
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