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UnknownPhase II Trial and Correlative Genomic Analysis of Everolimus Plus Bevacizumab in Advanced Non-Clear Cell Renal Cell Carcinoma.
Not available
S-EPMC5791841 | biostudies-literature
UnknownBRAF mutation and CDKN2A deletion define a clinically distinct subgroup of childhood secondary high-grade glioma.
Not available
S-EPMC4356711 | biostudies-literature
UnknownComprehensive and Equitable Care for Vulnerable Veterans With Integrated Palliative, Psychology, and Oncology Care.
Not available
S-EPMC8560117 | biostudies-literature
MetabolomicsNQO1 protects obese mice through improvements in glucose and lipid metabolism.
Chronic nutrient excess leads to metabolic disorders and insulin resistance. Activation of stress-responsive pathways via Nrf2 activation contributes to energy metabolism regulation. Here, inducible activation of Nrf2 in mice and transgenesis of the Nrf2 target, NQO1, conferred protection from diet-...
2024-05-23 | MTBLS2085 | MetaboLights
UnknownClinical and Genomic Characterization of Treatment-Emergent Small-Cell Neuroendocrine Prostate Cancer: A Multi-institutional Prospective Study.
Not available
S-EPMC6366813 | biostudies-literature
ProteomicsMitochondrial Protein Hyperacetylation in the Failing Heart
Mitochondrial proteomics was used to identify energy metabolic derangements that occur during the early stages of heart failure in well-defined mouse models. Levels of β-hydroxybutyrate dehydrogenase 1 (BDH1), a key enzyme in ketone oxidation, was upregulated. 13C-substrate flux studies and metabol...
ORGANISM(S): Mus musculus (Mouse) 
2016-02-26 | PXD001652 | Pride
Heart failure Acetylation Mitochondria 
UnknownSelective Inhibition of Nuclear Export With Oral Selinexor for Treatment of Relapsed or Refractory Multiple Myeloma.
Not available
S-EPMC6905485 | biostudies-literature
UnknownLoss of the long form of Plod2 phenocopies contractures of Bruck syndrome-osteogenesis imperfecta.
Not available
S-EPMC11371901 | biostudies-literature
ModelsVinnakotta2010_TranscientAnoxia_SOLmuscle
This a model from the article: Common phenotype of resting mouse extensor digitorum longus and soleus muscles: equal ATPase and glycolytic flux during transient anoxia. Vinnakota KC, Rusk J, Palmer L, Shankland E, Kushmerick MJ. J Physiol 2010 Jun 1;58...
2005-01-01 | MODEL1006230112 | BioModels
ModelsVinnakota2010_TranscientAnoia_EDLmuscle
This a model from the article: Common phenotype of resting mouse extensor digitorum longus and soleus muscles: equal ATPase and glycolytic flux during transient anoxia. Vinnakota KC, Rusk J, Palmer L, Shankland E, Kushmerick MJ. J Physiol 2010 Jun 1;58...
2005-01-01 | MODEL1006230100 | BioModels
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