Loss of LdtJ in Acinetobacter baumannii disrupts cell morphology, downregulates peptidoglycan precursor genes (e.g., dadA, alr), and activates the stringent response, including elevated ppGpp levels and dksA upregulation. These defects are fully suppressed in a ∆ldtJ ∆mla double mutant, implicating ...
ORGANISM(S): Acinetobacter baumannii