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ProteomicsEvolution and mechanism of MEIS2-mediated forelimb specialization in bats
The genomic basis of limb adaptations in tetrapods is thought to be largely driven by changes in gene regulation. However, the mechanisms by which regulatory programs evolve are not well understood. In bats, wing membrane development has been shown to be associated with expression of the transcripti...
ORGANISM(S): Mus musculus (Mouse) Carollia perspicillata 
2026-06-10 | PXD074776 | Pride
UnknownPreformed chromatin topology assists transcriptional robustness of Shh during limb development.
Not available
S-EPMC6589666 | biostudies-literature
UnknownRepression and 3D-restructuring resolves regulatory conflicts in evolutionarily rearranged genomes.
Not available
S-EPMC9567273 | biostudies-literature
UnknownEnhancer adoption by an LTR retrotransposon generates viral-like particles, causing developmental limb phenotypes.
Not available
S-EPMC12283350 | biostudies-literature
UnknownCombinatorial effects on gene expression at the Lbx1/Fgf8 locus resolve split-hand/foot malformation type 3.
Not available
S-EPMC10020157 | biostudies-literature
UnknownCleidocranial dysplasia: clinical and molecular genetics.
Not available
S-EPMC1734317 | biostudies-literature
TranscriptomicsDisruptions of Topological Chromatin Domains Causes Pathogenic Rewiring of Gene-Enhancer Interactions [4C-Seq-mouse]
Mammalian genomes are organized into megabase-scale topologically associated domains (TADs) that have been proposed to represent large regulatory units. Here we demonstrate that disruption of TADs can cause rewiring of long-range regulatory architecture and result in pathogenic phenotypes. We show t...
ORGANISM(S): Mus musculus 
2015-05-08 | E-GEOD-66379 | biostudies-arrayexpress
UnknownBMPs as new insulin sensitizers: enhanced glucose uptake in mature 3T3-L1 adipocytes via PPARγ and GLUT4 upregulation.
Not available
S-EPMC5722815 | biostudies-literature
UnknownGenetic disorders of the skeleton: a developmental approach.
Not available
S-EPMC1180673 | biostudies-literature
UnknownLgr5 and Col22a1 Mark Progenitor Cells in the Lineage toward Juvenile Articular Chondrocytes.
Not available
S-EPMC6829785 | biostudies-literature
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