Sort   by:  
 Page size 
ProteomicsIschemic preconditioning predominantly affects phosphosites involved in myocardial contraction and promotes myocardial stunning while reducing infarction size in rats.
Background: Ischemic preconditioning (IPC), i.e., brief periods of ischemia, protect the heart from subsequent prolonged ischemic injury, and reduces infarction size. Myocardial stunning refers to transient loss of contractility in the heart after myocardial ischemia that recovers without causing pe...
ORGANISM(S): Rattus norvegicus (Rat) 
2024-05-15 | PXD050441 | Pride
Myocardial infarction Phosphoproteomics Ischemic preconditioning Echocardiography Speckle tracking analysis Myocardial stunning 
ProteomicsComparison of the proteomic landscape in experimental ischemia-reperfusion with versus without ischemic preconditioning
Myocardial ischemic preconditioning (IPC) enhances myocardial resilience to ischemic injury. Myocardial stunning is a transient, reversible dysfunction, while necrosis involves irreversible cell death. The relationship between IPC, stunning, and necrosis is not well understood, requiring further mol...
ORGANISM(S): Rattus norvegicus (Rat) 
2025-04-09 | PXD050310 | Pride
Ischemia-reperfusion injury  tissue remodeling  therapeutic targets Myocardial ischemic preconditioning: myocardial stunning  myocardial infarction  cardiac proteomics  temporal changes 
TranscriptomicsIschaemic preconditioning blunted myocardial DNA methylation and regulated myocardial transcriptome in ischaemic reperfusion settings
Background. Ischemic preconditioning (IPC) reduces infarct size but may increase the extent of reversible cardiac dysfunction (myocardial stunning). DNA methylation is thought to regulate IPC-induced gene expression, impacting inflammation, oxidative stress, apoptosis, and DNA repair pathways. Aims....
ORGANISM(S): Rattus norvegicus 
2025-12-02 | GSE284606 | GEO
ProteomicsAnalysis of cardiac ischemia-reperfusion injury using DIA proteomics and N-terminomics
Data used in the publication: Analysis of cardiac ischemia-reperfusion injury using DIA proteomics and N-terminomics reveals alterations in mitochondrial function and metabolism
ORGANISM(S): Rattus (ncbitaxon:10114) 
MSV000092906 | MassIVE
Sort   by:  
 Page size