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ProteomicsBacterial pore-forming toxins remodel the host epithelial cell proteome
Bacterial pathogens use various strategies to interfere with host cell functions. Among these strategies, bacteria induce transcriptional changes, in order to modify the set of proteins synthetized by the host cell, or target pre-existing proteins by modulating their post-translational modifications...
ORGANISM(S): Homo sapiens (Human) 
2018-11-07 | PXD006900 | Pride
Listeria infection Pore-forming toxin Silac Listeriolysin o 
ProteomicsShort-term remodeling of the host epithelial cell proteome by the listeriolysin O pore-forming toxin
Bacterial pathogens use various strategies to interfere with host cell functions. Among these strategies, bacteria induce transcriptional changes, in order to modify the set of proteins synthetized by the host cell, or target pre-existing proteins by modulating their post-translational modifications...
ORGANISM(S): Homo sapiens (Human) 
2018-05-17 | PXD009339 | Pride
Pore-forming toxin Listeriolysin o Bacterial infection Listeria 
TranscriptomicsListeria monocytogenes induces T cell receptor unresponsiveness via its pore-forming toxin listeriolysin O
The success of many pathogens relies on their ability to circumvent the innate and adaptive immune defenses. How bacterial pathogens subvert host responses is not clear. Cholesterol-dependent cytolysins (CDCs) represent an expansive family of homologous pore-forming toxins produced by more than 20 G...
ORGANISM(S): Mus musculus 
2010-07-29 | E-GEOD-19337 | biostudies-arrayexpress
GenomicsLITAF PROMOTES MEMBRANE REPAIR AGAINST PORE-FORMING PROTEINS
LITAF PROMOTES MEMBRANE REPAIR AGAINST PORE-FORMING PROTEINS
PRJNA1015277 | ENA
ProteomicsA pore-forming toxin initiates ABI1 complex switching to promote bacterial cell-to-cell spread
Listeria monocytogenes (Lm), a prototypical intracellular bacterial pathogen, expresses ActA to initiate ARP2/3-mediated actin-based motility in the cytosol of host cells. Motile bacteria generate Lm-containing protrusions at the cell surface, facilitating direct cell-to-cell spread and disseminatio...
ORGANISM(S): Homo sapiens (Human) 
2026-06-15 | PXD074581 | Pride
Cell-to-cell spread Listeria Infectious diseases Ip-ms 
TranscriptomicsConserved immune regulator HLH-30/TFEB couples pore-forming toxin damage sensing to intestine homeostasis via TTC38 signal orchestrator
Effector-triggered immunity (ETI) safeguards intestinal epithelial cells (IECs) by surveilling bacterial virulence effectors and effector-induced cellular stress, such as plasma membrane damage induced by pore-forming toxins (PFTs). However, the knowledge about ETI regulators in maintaining IEC home...
ORGANISM(S): Caenorhabditis elegans 
2025-11-11 | E-MTAB-15698 | biostudies-arrayexpress
ProteomicsBacterial pore-forming toxins remodel the host epithelial cell proteome
Bacterial pathogens use various strategies to interfere with host cell functions. Among these strategies, bacteria induce transcriptional changes, in order to modify the set of proteins synthetized by the host cell, or target pre-existing proteins by modulating their post-translational modifications...
ORGANISM(S): Homo sapiens (Human) 
2018-11-07 | PXD006996 | Pride
Listeria Infection Listeriolysin o 
UnknownInhibition of Pore-Forming Proteins.
Not available
S-EPMC6784129 | biostudies-literature
GenomicsA widely-occurring family of pore-forming effectors broadens the impact of the Serratia Type VI secretion system
A widely-occurring family of pore-forming effectors broadens the impact of the Serratia Type VI secretion system
PRJNA1284335 | ENA
GenomicsThe pore-forming apolipoprotein APOL7C drives phagosomal rupture and antigen cross-presentation by dendritic cells
The pore-forming apolipoprotein APOL7C drives phagosomal rupture and antigen cross-presentation by dendritic cells
PRJNA1156795 | ENA
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