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TranscriptomicsTranscription profiling of human breast cancer cell lines and tissue samples to identify aberrant microRNA expression
MicroRNAs are a class of small non-coding RNAs that control gene expression by targeting messenger RNAs and triggering either translation repression or RNA degradation. Their aberrant expression may be involved in human diseases, including cancer. Indeed, microRNA aberrant expression has been previo...
ORGANISM(S): Homo sapiens 
2006-01-01 | E-TABM-23 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of human acute myeloid leukemia samples
AML
ORGANISM(S): Homo sapiens 
2010-05-10 | E-TABM-972 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of human ovarian carcinoma samples
OVARIAN EPITHELIAL CARCINOMA
ORGANISM(S): Homo sapiens 
2010-05-10 | E-TABM-975 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of human chronic lymphocytic leukemia samples
CLL
ORGANISM(S): Homo sapiens 
2010-05-10 | E-TABM-973 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of human acute myeloid leukemia samples
AML
ORGANISM(S): Homo sapiens 
2010-05-18 | E-TABM-974 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of human normal tissues
Normal Tissues
ORGANISM(S): Homo sapiens 
2010-05-18 | E-TABM-970 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of human normal tissues
Normal Tissues
ORGANISM(S): Homo sapiens 
2010-05-10 | E-TABM-969 | biostudies-arrayexpress
TranscriptomicsMicroRNA profiling by array of human metastatic breast cancer
metastatic breast cancer miRNA
ORGANISM(S): Homo sapiens 
2010-06-02 | E-TABM-971 | biostudies-arrayexpress
GenomicsThe immunopathological landscape of human pre-TCRα deficiency: from rare to common variants
The immunopathological landscape of human pre-TCRα deficiency: from rare to common variants
PRJNA1020588 | ENA
TranscriptomicsIn vitro responses of PBMC and fibroblasts from patients with TRIF deficiency after TRIF dependent and independent stimuli
We report here unrelated HSE patients with autosomal recessive (AR) or autosomal dominant (AD) TRIF deficiency. The AR form of the disease is due to a homozygous nonsense mutation, resulting in a complete absence of the TRIF protein. Both the TLR3 and the TRIF-dependent TLR4 signaling pathways are a...
ORGANISM(S): Homo sapiens 
2011-10-19 | E-GEOD-32390 | biostudies-arrayexpress
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