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TranscriptomicsTranscription profiling of Arabidopsis wild type, FIE- and MIA- loss-of-function mutants
Plants with genotypes wild type, FIE loss-of-function and MIA loss-of-function were compared. Plants were grown in growth chambers at 70% humidity and daily cycles of 16 h light and 8 h darkness at 21 C. Plant material used for the experiments was pooled from 10 plants. Siliques without withering fl...
ORGANISM(S): Arabidopsis thaliana 
2007-01-18 | E-ATMX-10 | biostudies-arrayexpress
TranscriptomicsAnti-proliferative tumor suppressor-like activity of mir-9 overexpression and identification of novel mir-9 targets by trancription profiling in breast cancer cells
Investigation of genome-wide effects (transcriptome profiling) and direct targets of mir-9 in MCF-7 cells transfected with mir-9 in two dosages (1X and 2X) Following transfections of mir-9 or control miRNA mimics, total RNA was isolated and microarray profiling was performed.
ORGANISM(S): Homo sapiens 
2012-09-04 | E-GEOD-33952 | biostudies-arrayexpress
TranscriptomicsBulk segregant sequencing (Bulk-Seq) analysis to identify paternal suppressors of medea in Arabidopsis thaliana
Arabidopsis thaliana seeds that maternally inherit a medea (mea) mutant allele abort before completing embryogenesis. However, mea seeds can be rescued by pollen from several natural ecotypes of A. thaliana, including the Cape Verdian accession Cvi-0. We developed a method for the mapping of parent-...
ORGANISM(S): Arabidopsis thaliana 
2016-11-18 | E-MTAB-5196 | biostudies-arrayexpress
TranscriptomicsTranscriptome profiling of an autopolyploidy series of Arabidopsis thaliana
Heterosis occurs where F1 offspring display superior characteristics to the parents. Heterosis is usually considered to result from crosses of genetically distinct (e.g. homozygous inbred) parents producing heterozygous F1 offspring. Most mechanistic models for heterosis require genetically heterozy...
ORGANISM(S): Arabidopsis thaliana 
2014-08-01 | E-GEOD-52885 | biostudies-arrayexpress
Transcriptomicsexpression data of miR-21 knockdown in MCF-7
miR-21 is overexpressed in breast cancer cells. Knock-down of miR-21 cause apoptosis and decreased cell proliferation. To identify miR-21 regulated cancer-pathways in MCF-7 cells, we knocked-down miR-21 and performed microarray. RNA was harvested from MCF-7 cells 24 hours transfection of anti-contro...
ORGANISM(S): Homo sapiens 
2014-03-01 | E-GEOD-52674 | biostudies-arrayexpress
UnknownTocilizumab in patients admitted to hospital with COVID-19 (RECOVERY): a randomised, controlled, open-label, platform trial.
Not available
S-EPMC8084355 | biostudies-literature
UnknownAzithromycin in patients admitted to hospital with COVID-19 (RECOVERY): a randomised, controlled, open-label, platform trial.
Not available
S-EPMC7884931 | biostudies-literature
UnknownConvalescent plasma in patients admitted to hospital with COVID-19 (RECOVERY): a randomised controlled, open-label, platform trial.
Not available
S-EPMC8121538 | biostudies-literature
UnknownAspirin in patients admitted to hospital with COVID-19 (RECOVERY): a randomised, controlled, open-label, platform trial.
Not available
S-EPMC8598213 | biostudies-literature
UnknownCasirivimab and imdevimab in patients admitted to hospital with COVID-19 (RECOVERY): a randomised, controlled, open-label, platform trial.
Not available
S-EPMC8830904 | biostudies-literature
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