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UnknownTranscriptional profiling identifies an androgen receptor activity-low, stemness program associated with enzalutamide resistance.
Not available
S-EPMC7275746 | biostudies-literature
UnknownPredicting phenotype and emerging strains among Chlamydia trachomatis infections.
Not available
S-EPMC2819883 | biostudies-literature
UnknownTranscriptional profiling of primary prostate tumor in metastatic hormone-sensitive prostate cancer and association with clinical outcomes: correlative analysis of the E3805 CHAARTED trial.
Not available
S-EPMC8463957 | biostudies-literature
UnknownSynthesis and evaluation of 2,5-substituted pyrimidines as small-molecule gankyrin binders.
Not available
S-EPMC10853842 | biostudies-literature
UnknownMetastasis-directed radiotherapy (MDRT) for men with de novo, oligometastatic prostate cancer treated with long-term androgen deprivation therapy in the STAMPEDE trial (METANOVA): protocol for a multicentre, phase II randomised controlled trial in the USA
Not available
S-EPMC13646922 | biostudies-literature
UnknownExploring the liver microenvironment following successful therapy for HCV: gene expression profiling and residual T cell infiltration.
Not available
S-EPMC12640901 | biostudies-literature
MetabolomicsObesogenic high-fat diet and MYC cooperate to promote lactate accumulation and tumor microenvironment remodeling in prostate cancer (Murine prostate assays)

Cancer cells exhibit metabolic plasticity to meet oncogene-driven dependencies while coping with nutrient availability. A better understanding of how systemic metabolism impacts the accumulation of metabolites that reprogram the tumor microenvironment and drive cancer could facilitate development...

2024-04-10 | MTBLS3317 | MetaboLights
UnknownBET Bromodomain Inhibition Blocks an AR-Repressed, E2F1-Activated Treatment-Emergent Neuroendocrine Prostate Cancer Lineage Plasticity Program.
Not available
S-EPMC8416959 | biostudies-literature
TranscriptomicsPI3K inhibition results in enhanced estrogen receptor function and dependence in hormone receptor-positive breast cancer
Activating mutations of PIK3CA are the most frequent genomic alterations in estrogen receptor (ER)-positive breast tumors and selective PI3Kα inhibitors are in clinical development. The activity of these agents, however, is not homogenous and only a fraction of patients bearing PIK3CA-mutant ER-posi...
ORGANISM(S): Homo sapiens 
2015-04-29 | E-GEOD-64033 | biostudies-arrayexpress
UnknownHeterogeneity in intrahepatic macrophage populations and druggable target expression in patients with steatotic liver disease-related fibrosis.
Not available
S-EPMC10757256 | biostudies-literature
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