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ProteomicsQuantification of sulfenic acid on model protein
Aldolase contains eight free cysteine residues without disulfide formation in its native state, which makes it a suitable model to manipulate the oxidative modifications on cysteine. H2O2 was chosen to treat aldolase since the principle product between which is sulfenic acid.To assess the reduction ...
ORGANISM(S): Lagomorpha Mammalia Gallus gallus (Chicken) 
2016-10-10 | PXD003348 | Pride
Lysozyme Sulfenic acid Aldolase Arsenite 
ProteomicsQuantification of sulfenic acid modifications on an atherosclerotic model-1
Redox imbalance in cells, due to the accumulation of reactive oxygen species (ROS), has been implicated in the pathogenesis of several diseases, including atherosclerosis. However, the molecular details of redox regulation in atherosclerosis remain to be established.1 During the progression of ather...
ORGANISM(S): Homo sapiens (Human) 
2016-10-10 | PXD003354 | Pride
Atheroscelrosis Sulfenic acid Biotin switch assay 
ProteomicsQuantification of sulfenic acid modification in an atherosclerosis
Physiological stimuli such as thrombin, or pathological stimuli such as lysophosphatidic acid (LPA), activate platelets. The activated platelets bind to monocytes through P-selectin-PSGL-1 interactions, but also release the contents of their granules, commonly called “platelet releasate”. It is know...
ORGANISM(S): Homo sapiens (Human) 
2016-10-10 | PXD004862 | Pride
Sulfenic acid Biotin switch assay Thp-1 cell Nox 
ProteomicsQuantification of sulfenic acid modifications in an atherosclerotic model
Physiological stimuli such as thrombin or pathological stimuli such as lysophosphatidic acid (LPA) activate platelets. The activated platelets bind to monocyte through P-selectin-PSGL-1 interactions, but also release the contents of their granules, which were defined as platelet releasate. It is kno...
ORGANISM(S): Homo sapiens (Human) 
2016-10-10 | PXD003365 | Pride
Sulfenic acid Biotin switch assay Atherosclerosis 
ProteomicsQuantification of sulfenic acid modifications in an atherosclerotic model-2
Physiological stimuli such as thrombin or pathological stimuli such as lysophosphatidic acid (LPA) activate platelets. The activated platelets bind to monocyte through P-selectin-PSGL-1 interactions, but also release the contents of their granules, which were defined as platelet releasate. It is kno...
ORGANISM(S): Homo sapiens (Human) 
2016-10-10 | PXD003363 | Pride
Sulfenic acid Biotin switch assay Atherosclerosis 
UnknownSulfenic acid chemistry, detection and cellular lifetime.
Not available
S-EPMC4184475 | biostudies-literature
UnknownLight-Mediated Sulfenic Acid Generation from Photocaged Cysteine Sulfoxide.
Not available
S-EPMC4699549 | biostudies-literature
UnknownWidespread sulfenic acid formation in tissues in response to hydrogen peroxide.
Not available
S-EPMC539740 | biostudies-literature
UnknownThe reversible formation of cysteine sulfenic acid promotes B-cell activation and proliferation.
Not available
S-EPMC3659792 | biostudies-literature
ProteomicsWittig reagents for chemoselective sulfenic acid ligation enables global site stoichiometry analysis and redox-controlled mitochondrial targeting
Triphenylphosphonium (TPP) ylides, known as Wittig reagents, are one of the most commonly used tools in synthetic chemistry. Despite their considerable versatility, Wittig reagents have not yet been explored for their utility in biological applications. Here, we introduce a new chemoselective ligati...
ORGANISM(S): Homo Sapiens 
2021-04-26 | PXD025630 |
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