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TranscriptomicsTranscription profiling of QUB HCT116 sensitive and resistant colorectal cancer time series
HCT116 parental, HCT116 5-FU resistant and HCT116 oxaliplatin resistant cells have been transiently treated with with their respective drug (5-FU or oxaliplatin) for 0, 6 12 or 24h in 3 independent experiments.
ORGANISM(S): Homo sapiens 
2006-01-01 | E-MEXP-390 | biostudies-arrayexpress
TranscriptomicsMethylation profiling by array of human cell line HCT116 after treatment with demethylating drugs azacytidine or decitabine.
array-based analysis of genome-wide DNA methylation changes induced by the demethylating drugs azacytidine and decitabine on HCT116 cells for 24 hours
ORGANISM(S): Homo sapiens 
2010-07-01 | E-MTAB-210 | biostudies-arrayexpress
Transcriptomicsp53 shapes genome-wide changes in small non-coding RNA expression during the human DNA damage response
Small RNA-seq on MCF10A, HCT116 and HCT116p53-/- cell lines after induction of DNA damage (5 Gy Irradiation). Small RNA-seq on MCF10A, HCT116 and HCT116p53-/- at 4 and 24 hours after induction of DNA damage (5 Gy Irradiation), done in duplicate with respective control (0 hour) using illumina Genome ...
ORGANISM(S): Homo sapiens 
2014-08-15 | E-GEOD-50064 | biostudies-arrayexpress
TranscriptomicsMethylation profiling by array of human HCT116 and HL60 cells treated with the demthylating drugs azacytidine and decitabine
Array-based analysis of genome-wide DNA methylation changes induced by the demethylating drugs azacytidine and decitabine on HCT116 and HL60 cells for 24 hours
ORGANISM(S): Homo sapiens 
2010-12-01 | E-MTAB-417 | biostudies-arrayexpress
TranscriptomicsMutant p53R273H regulated microRNA profiling in presence and absence of DNA damage in H1299 cells
In the present study, we used a high-throughput small RNA deep sequencing followed by a systematic computational analysis to identify genome wide mutant p53R273H regulated miRNAs in both DNA damage dependent and independent context. Several miRNA-mRNA regulatory networks have been predicted that mig...
ORGANISM(S): Homo sapiens 
2017-04-28 | E-GEOD-68353 | biostudies-arrayexpress
Genomicsena-DATASET-JHKC-27-11-2012-16:13:56:837-3 - samples
Neuroblastomas are tumors of peripheral sympathetic neurons and are the most common solid tumor in children. To determine the genetic basis for neuroblastoma we performed whole-genome sequencing (6 cases), exome sequencing (16 cases), genome-wide rearrangement analyses (32 cases), and targeted ana...
EGAD00001000282 | EGA
GenomicsIntegrated genomic analyses identify ARID1A and ARID1B alterations in the childhood cancer neuroblastoma
Neuroblastomas are tumors of peripheral sympathetic neurons and are the most common solid tumor in children. To determine the genetic basis for neuroblastoma we performed whole-genome sequencing (6 cases), exome sequencing (16 cases), genome-wide rearrangement analyses (32 cases), and targeted ana...
EGAS00001000369 | EGA
GenomicsNeuroblastoma Data Access Committee
Data Access Committee EGAC00001000085
EGAC00001000085 | EGA
Genomicsena-DATASET-MSKCC-27-01-2017-19:20:42:018-272 - samples
Sequencing of untreated pancreatic cancer metastases and primary tumor sections.
EGAD00001003153 | EGA
GenomicsSequencing of pancreatic cancer primary tumors and metastases
The extent of heterogeneity of driver gene mutations present in naturally occurring metastases is largely unknown, i.e. treatment-naïve metastatic disease. To address this issue, 60x whole genome sequencing of 26 metastases from 4 patients was carried out. We found that the identical driver gene m...
EGAS00001002186 | EGA
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