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GenomicsChIP-seq of sigma 70 in wild-type and hns mutant Escherichia coli K-12 MG1655
We mapped the genome-wide binding of sigma 70 in E. coli K-12 MG1655 and an hns mutant that is otherwise isogenic using ChIP coupled with deep sequencing (ChIP-seq). We show that intragenic binding of sigma 70 is increased in the hns mutant.
ORGANISM(S): Escherichia coli 
2013-12-15 | E-MTAB-2148 | biostudies-arrayexpress
TranscriptomicsSNP data from human fibroblasts and induced Pluripotent Stem cells from Parkinson's Disease patients
We have generated human induced Pluripotent Stem cells (hiPSc) from Parkinson's Disease patients, using retrovirus-mediated delivery of reprogramming factors. hiPSc lines have been screened using SNP array to assess chromosomal stability (alongside the fibroblast lines from which they derived), and ...
ORGANISM(S): Homo sapiens 
2013-12-18 | E-GEOD-53425 | biostudies-arrayexpress
TranscriptomicsRNA-seq of MG1655 wt and MG1655 delta-phoB strains grown in MOPS media with low phosphate (0.2 mM)
We used RNA-seq to determine differences in RNA levels between wild-type E. coli K-12 and a ΔphoB mutant, each grown in MOPS media with low phosphate (0.2 mM).
ORGANISM(S): Escherichia coli str. K-12 substr. MG1655 
2020-07-12 | E-MTAB-9292 | biostudies-arrayexpress
TranscriptomicsMG1655 araC arabinose transcription profiling
AraC is an Escherichia coli transcription factor that regulates genes in response to the presence/absence of arabinose. We used transcription profiling to determine RNA levels in Escherichia coli K-12 strain MG1655 and MG1655 delta araC grown either in the absence or the presence of arabinose. Thus,...
ORGANISM(S): Escherichia coli K-12 
2013-10-19 | E-MTAB-1916 | biostudies-arrayexpress
TranscriptomicsChIP-seq of MG1655 wt, MG1655 ΔphoB, MG1655 Δhns, MG1655 ΔphoB+Δhns and MG1655 PhoB-FLAG strains grown in MOPS media with either low phosphate (0.2 mM) or high phosphate (1.32 mM)
We used ChIP-seq to map the binding of of C-terminally FLAG3-tagged PhoB to the Escherichia coli K-12 genome during growth in MOPS minimal media with low phosphate or high phosphate (0.2 mM or 1.32 mM, respectively). As a control, we performed ChIP-seq in an untagged strain. We also used ChIP-seq to...
ORGANISM(S): Escherichia coli str. K-12 substr. MG1655 
2020-07-12 | E-MTAB-9293 | biostudies-arrayexpress
TranscriptomicsRNA-seq of MG1655 wt and MG1655 delta-phoB strains grown in MOPS media with low phosphate (0.2 mM)
We used RNA-seq to determine differences in RNA levels between wild-type E. coli K-12 and a ΔphoB mutant, each grown in MOPS media with low phosphate (0.2 mM).
ORGANISM(S): Escherichia coli O17 
2020-10-03 | E-MTAB-9591 | biostudies-arrayexpress
GenomicsMG1655 flagellar regulator ChIP-seq (FlhD, FlhC, FliA)
We mapped the genome-wide binding of the flagellar regulators FlhD, FlhC, and FliA in FLAG-tagged derivatives of E. coli K-12 MG1655 using ChIP coupled with deep sequencing (ChIP-seq). We identify new binding sites for each factor.
ORGANISM(S): Escherichia coli 
2014-04-10 | E-MTAB-2457 | biostudies-arrayexpress
GenomicsSNP data from human fibroblasts and induced Pluripotent Stem cells from Parkinson's Disease patients
We have generated human induced Pluripotent Stem cells (hiPSc) from Parkinson's Disease patients, using retrovirus-mediated delivery of reprogramming factors. hiPSc lines have been screened using SNP array to assess chromosomal stability (alongside the fibroblast lines from which they derived), and ...
ORGANISM(S): Homo sapiens 
2013-12-18 | GSE53425 | GEO
TranscriptomicsMG1655 Flagellar Regulators RNA-seq
This experiment contains RNA-seq data for a motile derivative of MG1655 and isogenic strains with deletions of each flagellar regulator: flhD, flhC, and fliA. All strains were grown at 37 degrees C in LB. After RNA purification, ribosomal RNA was removed using RiboZero and a library was made of rema...
ORGANISM(S): Escherichia coli str. K-12 substr. MG1655 
2014-08-01 | E-MTAB-2802 | biostudies-arrayexpress
UnknownCost-Effectiveness of Chimeric Antigen Receptor T Cell Therapy in Patients with Relapsed or Refractory Large B Cell Lymphoma: No Impact of Site of Care.
Not available
S-EPMC9309131 | biostudies-literature
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