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TranscriptomicsElevated expression of AKR1C3 leads esophageal cancer resistant to ionizing radiation via modulation of oxidative stress (TE13)
With the aim to elucidate the etiology of radioresistance, we explored the genetic alterations in non-radioresistant vs. resistant esophageal cancer cells acquired by long-term fractionated radiation. We found AKR1C3, an aldo-keto reductase expressed seldom in most human tissues, expressed higher in...
ORGANISM(S): Homo sapiens 
2014-09-27 | E-GEOD-61816 | biostudies-arrayexpress
TranscriptomicsElevated expression of AKR1C3 leads esophageal cancer resistant to ionizing radiation via modulation of oxidative stress
With the aim to elucidate the etiology of radioresistance, we explored the genetic alterations in non-radioresistant vs. resistant esophageal cancer cells acquired by long-term fractionated radiation. We found AKR1C3, an aldo-keto reductase expressed seldom in most human tissues, expressed higher in...
ORGANISM(S): Homo sapiens 
2014-09-27 | E-GEOD-61620 | biostudies-arrayexpress
TranscriptomicsElevated expression of AKR1C3 leads esophageal cancer resistant to ionizing radiation via modulation of oxidative stress (KY170)
With the aim to elucidate the etiology of radioresistance, we explored the genetic alterations in non-radioresistant vs. resistant esophageal cancer cells acquired by long-term fractionated radiation. We found AKR1C3, an aldo-keto reductase expressed seldom in most human tissues, expressed higher in...
ORGANISM(S): Homo sapiens 
2014-09-27 | E-GEOD-61772 | biostudies-arrayexpress
UnknownCause of Death and Predictors of All-Cause Mortality in Anticoagulated Patients With Nonvalvular Atrial Fibrillation: Data From ROCKET AF.
Not available
S-EPMC4943233 | biostudies-literature
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