Epstein Barr Virus Nuclear Antigen 3C binds to BATF/IRF4/SPI1 sites and represses CDKN2A by recruiting Sin3A

ABSTRACT: We report the application of ChIP Seq to study the Epstein Barr Virus Nuclear Antigen 3C, an essential transcriptional regulator involved in the transformation of Resting B Lymphocytes to the immortalized Lymphoblast Cell Lines Examination of viral and cellular transcription factors in 1 type of cell line

ORGANISM(S): Homo sapiens

SUBMITTER: Hufeng Zhou 

PROVIDER: E-GEOD-52632 | biostudies-arrayexpress |

REPOSITORIES: biostudies-arrayexpress