Synergism between PPAR? and glucocorticoid receptor signaling promotes self-renewal of BFU-E erythroid progenitors and increases red cell production [RNA-seq]

ABSTRACT: Analyses of gene expression by RNA-Seq in mouse E14.5 fetal liver burst-forming unit erythroid (BFU-E) cells untreated or treated by dexamethasone (DEX) with or without PPAR? agonist GW7647. RNA-Seq was performed on enriched populations of mouse BFU-E isolated from E14.5 fetal liver, as well as BFU-E enriched cells treated with Dex ± GW7647.

ORGANISM(S): Mus musculus

SUBMITTER: Xiaofei Gao 

PROVIDER: E-GEOD-63836 | biostudies-arrayexpress |

REPOSITORIES: biostudies-arrayexpress