Expression data of murine model of cardiac hypertrophy

ABSTRACT: An initial cellular change in the pathogenesis of heart failure is cardiomyocyte hypertrophy, characterized by increased cell size, enhanced protein synthesis and reactivation of fetal genes. In addition to mechanical stresses, several neurohumoral factors have been identified as potent hypertrophic agents, including angiotensin II, endothelin, and catecholamines. We used microarrays to study the gene expression during cardiac hypertrophy.

ORGANISM(S): Mus musculus

PROVIDER: GSE26671 | GEO | 2011/12/31

SECONDARY ACCESSION(S): PRJNA136419

REPOSITORIES: GEO