Genomics

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Expression data from Hfe-deficient liver and duodenum in mouse strains with differing susceptibilities to iron loading


ABSTRACT: Hfe disruption in the mouse leads to experimental hemochromatosis by a mechanism which remains elusive. Evidence for at least five modifier genes has been obtained. These account for the higher iron load of Hfe-deficient D2 mice compared to B6 mice. Gene expression profling was used to clarify the mechanism of Hfe action and to identify potential modifier genes. Keywords: response to genetic modification

ORGANISM(S): Mus musculus

PROVIDER: GSE7357 | GEO | 2008/03/22

SECONDARY ACCESSION(S): PRJNA100429

REPOSITORIES: GEO

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