Exploring the mechanism of contact-dependent cell-cell communication on chemoresistance based on single-cell high-throughput drug screening platform
Ontology highlight
ABSTRACT:
INSTRUMENT(S): Q Exactive HF
ORGANISM(S): Homo Sapiens (human)
SUBMITTER: 
He Huang
LAB HEAD: He Huang
PROVIDER: PXD047031 | Pride | 2025-05-06
REPOSITORIES: Pride
Dataset's files
| Action | DRS | |||
|---|---|---|---|---|
| CPTX_proteinGroups.tsv | Tabular | |||
| CP_proteinGroups.tsv | Tabular | |||
| PTX_proteinGroups.tsv | Tabular | |||
| cellcommunity_CPTX_re1.mzXML | Mzxml | |||
| cellcommunity_CPTX_re2.mzXML | Mzxml |
Items per page: 5 1 - 5 of 22 |
Publications
SIRT3 differentially regulates lysine benzoylation from SIRT2 in mammalian cells.
Peng Panpan P Lu Ying Y Ren Xuelian X Yan Cong C Guo Xinlong X Liu Ruilong R Song Xiaohan X Huang He H
iScience 20241016 11
Lysine benzoylation (Kbz), a new type of protein post-translational modification (PTM) we discovered, has garnered significant attention. While we initially identified SIRT2 as a debenzoylase in mammalian cells, recent findings suggest its exclusivity may be questioned. However, other debenzoylases in mammalian cells remain underexplored. Here, our study reveals SIRT3 as an additional debenzoylase. Through quantitative analysis, we identified 1,075 Kbz sites in mammalian cells, with 44 specifica ...[more]
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