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Mus musculus strain:Lewis Lung Carcinoma Cell Line, P29

ABSTRACT: Paradigm conversion of mammalian mitochondrial genome by transmitochondrion (A)

PROVIDER: PRJDB3861 | ENA |

REPOSITORIES: ENA

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			Action	DRS
	DRR044993_1.fastq.gz	Fastqsanger.gz
	DRR044993_2.fastq.gz	Fastqsanger.gz
	DRR044994_1.fastq.gz	Fastqsanger.gz
	DRR044994_2.fastq.gz	Fastqsanger.gz
	DRR044995_1.fastq.gz	Fastqsanger.gz

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Project description:Radiotherapy is one of the most common therapies for cancer. Approximately half of all cancer patients will receive radiotherapy at some point during treatment. Consequences of IR treatment are dose dependent and different sensitivity to IR of various types of cells is well established. To reduce the damage of IR to most sensitive cells of normal (noncancerous) tissue radiotherapy is administered as fractionated dose treatment applying radiation in ~2 Gy fractions every 24 hours, 5 times per week. However, during the therapy intrinsic and acquired tumor radioresistance may result in treatment failures. Comprehensive mechanisms of the resistance to irradiation as well as mechanisms of cellular response to fractionated dose IR remain unclear. Therefore, in the present study we evaluated global gene expression changes in murine Lewis lung carcinoma LLC1 cells following X-ray irradiation of single 2 Gy or 10 Gy and 2 Gy x 5 fractionated doses.

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