Browse
Submit Data
Databases
API
Help

Dataset Information

0 Views

0 Connections

0 Citations

0 Reanalyses

0 Downloads

Omics score: 0

Differentially expressed mRNA in biological premature ovarian insufficient (bPOI)

ABSTRACT: Differentially expressed mRNA in biological premature ovarian insufficient (bPOI)

PROVIDER: PRJNA1260993 | ENA |

REPOSITORIES: ENA

ACCESS DATA

Json Xml

Dataset's files

Source:

			Action	DRS
	SRR33483448_1.fastq.gz	Fastqsanger.gz
	SRR33483448_2.fastq.gz	Fastqsanger.gz
	SRR33483449_1.fastq.gz	Fastqsanger.gz
	SRR33483449_2.fastq.gz	Fastqsanger.gz
	SRR33483450_1.fastq.gz	Fastqsanger.gz

Items per page:

1 - 5 of 34

Similar Datasets

Differentially expressed lncRNA in biological premature ovarian insufficient(bPOI)

Project description:Differentially expressed lncRNA in biological premature ovarian insufficient(bPOI)

| PRJNA665514 | ENA

Differentially expressed mRNA in biological premature ovarian insufficiency (bPOI)

Project description:Differentially expressed mRNA in biological premature ovarian insufficiency (bPOI)

| PRJNA830696 | ENA

Differentially expressed lncRNA in biological premature ovarian insufficiency (bPOI)

Project description:Differentially expressed lncRNA in biological premature ovarian insufficiency (bPOI)

| PRJNA559738 | ENA

Differentially expressed miRNA in biological premature ovarian insufficiency (bPOI)

Project description:We first examined changes of miRNAs and expression using microarrays ( Exiqon miRCURY™ microarray LNA v.18.0) of GCs from 10 bPOI and 10 age and BMI matched normal control women. Seventy-five miRNAs (Fold Change [FC]>2, P<0.05) were identified differentially expressed between bPOI patients and control women.

2018-04-11 | GSE100238 | GEO

Differentially expressed lncRNA in biological premature ovarian insufficiency (bPOI)

Project description:Our study is designed to demonstrate altered profiles of lncRNAs in the granulosa cells from patients with bPOI. Functional studies will be further confirmed. This study will provide new viewpoint for understanding the roles of non-coding RNA for GCs function, as well as potential etiologic mechanism for POI.

2020-02-17 | GSE135697 | GEO

Differentially expressed lncRNA in biological premature ovarian insufficient(bPOI)

Project description:We have completed the lncRNA expression profiling of human granulosa cells to reveals the differentially expressed lncRNAs involved in POI.

2021-02-20 | GSE158526 | GEO

Differentially expressed mRNA in biological premature ovarian insufficient (bPOI)

Project description:We have completed the mRNA expression profiling of human granulosa cells to reveals the differentially expressed mRNAs involved in POI.

2025-05-13 | GSE296744 | GEO

Differentially expressed mRNA in biological premature ovarian insufficiency (bPOI)

Project description:The aim of this study is to identify the expression profile of mRNA in granulosa cells of POI We performed gene expression profiling analysis using data obtained from RNA-seq of the GCs in 6 POI patients and 5 control patients.This study provides a better understanding of molecular mechanism of POI and identifies the possible biomarkers of POI.

2023-10-31 | GSE201276 | GEO

Homo sapiens

Project description:Differentially expressed miRNA in biological premature ovarian insufficiency (bPOI)

| PRJNA391142 | ENA

Homozygosity mapping in family segregating premature ovarian insufficiency (POI)

Project description:Premature ovarian insufficiency (POI) is defined as a primary ovarian defect characterized by absent menarche (primary amenorrhea) or premature depletion of ovarian follicles before the age of 40 (secondary amenorrhea) with hypergonadotropism and hypoestrogenism. Premature ovarian insufficiency has few known genetic causes but in familial cases a genetic link is often suspected. A large consanguineous family with three female affected with POI was investigated. All samples including 3 affected and 5 unaffecd underwent whole genome SNP genotyping using Affymetric Axiom_GW_Hu_SNP array. Linkage analysis was carried out using HomozygosityMapper and Allegro softwares.Linkage analysis mapped the disease phenotype to long arm of chromosome 20. Sequence data analysis of potential candidate genes failed to detect any pathogenic variant.

2014-03-20 | GSE56043 | GEO

OmicsDI is part of the ELIXIR infrastructure

OmicsDI is an Elixir interoperability service. Learn more ›

Tweets

OmicsDI Databases

PRIDE
PeptideAtlas
MassIVE
JPOST Repository
Physiome Model Repository

EGA
EVA
ENA
LINCS
PAXDB
Cell Collective

MetaboLights
Metabolomics Workbench
MetabolomeExpress
GNPS
BioModels
FAIRDOMHub

ArrayExpress
dbGaP
ExpressionAtlas
GEO
NODE

Information

Databases
Help
API
Contact us
Code on GitHub
Terms of use
Submit Data