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GenomicsAlkalibacter mobilis strain:M17 DMB
Alkalibacter sp. M17 DMB Genome sequencing and assembly
PRJNA676094 | ENA
Genomics
DMB-VierIDs
PRJEB75745 | ENA
UnknownTargeting human telomeric G-quadruplex DNA and inhibition of telomerase activity with [(dmb)2Ru(obip)Ru(dmb)2](4+).
Not available
S-EPMC3874006 | biostudies-literature
UnknownSalmonella enterica synthesizes 5,6-dimethylbenzimidazolyl-(DMB)-α-riboside. Why some Firmicutes do not require the canonical DMB activation system to synthesize adenosylcobalamin.
Not available
S-EPMC5218876 | biostudies-literature
UnknownHLA-DMB restricts human T-cell leukemia virus type-1 (HTLV-1) protein expression via regulation of ATG7 acetylation.
Not available
S-EPMC5663917 | biostudies-literature
UnknownBacillus megaterium has both a functional BluB protein required for DMB synthesis and a related flavoprotein that forms a stable radical species.
Not available
S-EPMC3573010 | biostudies-literature
UnknownPharmacokinetic Bioequivalence, Safety, and Immunogenicity of DMB-3111, a Trastuzumab Biosimilar, and Trastuzumab in Healthy Japanese Adult Males: Results of a Randomized Trial.
Not available
S-EPMC4746214 | biostudies-literature
UnknownRecreational athletes during downhill-mountain biking (DMB) show high incidence of upper extremity fractures in combination with soft-tissue injuries.
Not available
S-EPMC10879515 | biostudies-literature
TranscriptomicsTranscriptional Response of Zebrafish Embryos Exposed to Neurotoxic Compounds Reveals a Muscle Activity Dependent hspb11 Expression
Acetylcholinesterase (AChE) inhibitors are widely used as pesticides and drugs. Their primary effect is the overstimulation of muscular activity. Whether AChE inhibitors and related neurotoxic compound also provoke specific changes in gene transcription patterns that allow to establish a mechanistic...
ORGANISM(S): Danio rerio 
2011-12-01 | E-GEOD-27680 | biostudies-arrayexpress
ProteomicsProteomics of Mouse Heart Ventricles Reveals Mitochondria and Metabolism as Major Targets of a Post-Infarction Short-Acting GLP1Ra-Therapy Proteomics of Mouse Heart Ventricles Reveals Mitochondria and Metabolism as Major Targets of a Post-Infarction Short-Acting GLP1Ra-Therapy
Cardiovascular disease is the main cause of death worldwide, making it crucial to search for new therapies to mitigate major adverse cardiac events (MACEs) after a cardiac ischemic episode. Drugs in the class of the glucagon-like peptide-1 receptor agonists (GLP1Ra) have demonstrated benefits for he...
ORGANISM(S): Mus musculus (Mouse) 
2021-09-07 | PXD027867 | Pride
Mitochondrion  cellular respiration  metabolism Proteomics  glucagon-like peptide-1 receptor agonists  dmb  early cardiac remodeling 
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