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Transcriptomicsc-MET inhibitor inducing KSHV+ primary effusion lymphoma apoptosis
KSHV is a principal causative agent of primary effusion lymphoma (PEL). Despite this knowledge about the close relationship between HGF/c-MET network and solid tumors development, the role of HGF/c-MET in KSHV-related malignancies remains mostly unclear. We report that HGF/c-MET pathway is highly ac...
ORGANISM(S): Homo sapiens 
2016-01-05 | E-GEOD-70594 | biostudies-arrayexpress
TranscriptomicsDEN-induced HCC in c-Met knockout mice
HGF has been reported to have both positive and negative effects on carcinogenesis. Here we show that the loss of c-Met signaling in hepatocytes enhanced rather than suppressed the early stages of chemical hepatocarcinogenesis. c-Met conditional knockout mice (c-metfl/fl, AlbCre+/-; MetLivKO) treate...
ORGANISM(S): Mus musculus 
2008-08-09 | E-GEOD-8747 | biostudies-arrayexpress
UnknownCooperative interaction of MUC1 with the HGF/c-Met pathway during hepatocarcinogenesis.
Not available
S-EPMC3542123 | biostudies-literature
Unknown[Dysregulation of HGF/c-Met signal pathway and their targeting drugs in lung cancer].
Not available
S-EPMC6000362 | biostudies-literature
UnknownC7 peptide inhibits hepatocellular carcinoma metastasis by targeting the HGF/c-Met signaling pathway.
Not available
S-EPMC6804814 | biostudies-literature
UnknownPancreatic Cancer Treatment Targeting the HGF/c-MET Pathway: The MEK Inhibitor Trametinib.
Not available
S-EPMC10930669 | biostudies-literature
UnknownHGF/c-Met pathway facilitates the perineural invasion of pancreatic cancer by activating the mTOR/NGF axis.
Not available
S-EPMC9023560 | biostudies-literature
UnknownMolecular mechanism study of HGF/c-MET pathway activation and immune regulation for a tumor diagnosis model.
Not available
S-EPMC8278741 | biostudies-literature
UnknownImpairment of peripheral nerve regeneration by insufficient activation of the HGF/c-Met/c-Jun pathway in aged mice.
Not available
S-EPMC9647475 | biostudies-literature
ProteomicsTyrosine phosphorylation of IQGAP1 in the MET pathway
Mass spectrometry was used to determine IQGAP1 is a substrate of MET and c-Src, and is phosphorylated exclusively on Tyr-1510 under conditions with enhanced MET or c-Src cellular activity.
ORGANISM(S): Human 
MSV000086089 | MassIVE
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