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MetabolomicsNeuronal Hyperactivity due to Loss of Inhibitory Tone in APOE4 Mice Lacking Alzheimer’s Disease-like Pathology
Metabolite profiling was performed on metabolites extracted from the entorhinal cortex and primary visual cortex of 14-15 month old APOE3/3, APOE3/4 and APOE4/4 mice. Metabolites were run on a TOF Mass Spectrometer using an ANP column. Initial analysis was done in an untargeted manner, and processin...
2017-09-05 | MTBLS530 | MetaboLights
MetabolomicsEC and PVC from 14-15 month-old APOE3/3, APOE3/4 and APOE4/4 mice
Not available
ORGANISM(S): Mus musculus 
ST001819 | MetabolomicsWorkbench
MetabolomicsWT neurons treated with APOE3/3 and APOE4/4 ACM
Not available
ORGANISM(S): Mus musculus 
ST001820 | MetabolomicsWorkbench
MetabolomicsAPOE modulates microglial immunometabolism in response to age, amyloid pathology, and inflammatory challenge (Part 2 of 3)
Not available
ORGANISM(S): Mus musculus 
ST002451 | MetabolomicsWorkbench
MetabolomicsAPOE modulates microglial immunometabolism in response to age, amyloid pathology, and inflammatory challenge (Part 3 of 3)
Not available
ORGANISM(S): Mus musculus 
ST002453 | MetabolomicsWorkbench
GenomicsMus musculus strain:ApoE -/-
ApoE-/- atherosclerosis reversal
PRJNA530042 | ENA
MetabolomicsAPOE modulates microglial immunometabolism in response to age, amyloid pathology, and inflammatory challenge (Part 1 of 3)
Not available
ORGANISM(S): Mus musculus 
ST002450 | MetabolomicsWorkbench
ProteomicsRevisiting the relevance of the accumulation of apolipoprotein E (APOE) in mitochondria-associated ER membranes: Investi-gation of hepatic protein-protein interactions of APOE with LONP1, TOMM40 and VDAC1
Investigating the subcellular distribution and protein interaction of APOE and the modulation by cellular stress signals, we aimed to contribute to the elucidation of putative novel functions. We identified APOE as hepatic mitochondria-associated ER-membranes (MAM) protein, where it most likely inte...
ORGANISM(S): Homo sapiens (Human) 
2025-05-07 | PXD055281 | Pride
Mitochondria-er contacts (merc) Mam Stress response Proteome analysis Subcellular distribution Liver Apoe4 
TranscriptomicsGene Expression of ApoE Null and ApoE Null/RAGE Diabetic and Non-diabetic Mice
The multi-ligand Receptor for AGE (RAGE) contributes to atherosclerosis in apolipoprotein (ApoE) null mice in both the non-diabetic and diabetic states. Previous studies using soluble RAGE, the extracellular ligand-binding domain of RAGE, or homozygous RAGE null mice showed that blockade or deletion...
ORGANISM(S): Mus musculus 
2010-03-07 | E-GEOD-15729 | biostudies-arrayexpress
TranscriptomicsGene expression profiling in thoracic aortas of 8 week and 16 week ApoE knockout mice on a western type diet
Identification of novel pathways in the development of atherosclerosis. Here, we are looking at changes in gene expression that occur in the aorta with the development of atherosclerosis Analysis used RNA from thoracic aortas from chow fed ApoE knockout mice as control samples for comparison to the ...
ORGANISM(S): Mus musculus 
2015-02-03 | E-GEOD-65494 | biostudies-arrayexpress
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