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Bmpr2 mutation in murine PMVEC

Pulmonary arterial hypertension (PAH) is thought to be driven by dysfunction of pulmonary vascular microendothelial cells (PMVEC). Most hereditary PAH is associated with BMPR2 mutations. However, the physiologic and molecular consequences of expression of BMPR2 mutations in PMVEC are unknown. PMVEC ...

ORGANISM(S): Mus musculus

2011-07-01 | E-GEOD-28043 | biostudies-arrayexpress

Effect of BMPR2 mutation on vascular smooth muscle gene expression

BMPR2 mutation is the cause of most hereditary pulmonary arterial hypertension, but the common molecular consequence of different types of BMPR2 mutation is still not known. The goal of this study was to determine the common molecular consequences of three different classes of patient-derived BMPR2 ...

ORGANISM(S): Rattus norvegicus

2010-08-25 | E-GEOD-21573 | biostudies-arrayexpress

SPE-IMS-MS: An automated platform for sub-sixty second surveillance of endogenous metabolites and xenobiotics in biofluids

Characterization of endogenous metabolites and xenobiotics is essential to deconvoluting the genetic and environmental causes of disease. However, surveillance of chemical exposure and disease-related changes in large cohorts requires an analytical platform that offers rapid measurement, high sensit...

2017-02-14 | MTBLS427 | MetaboLights

Genomewide association studies in ankylosing spondylitis

The aim of this study was to identify genes associated with ankylosing spondylitis susceptibility in British and Australian individuals of European descent.

EGAS00000000104 | EGA

WTCCC2 project samples from Ankylosing spondylitis Cohort

EGAD00010000150 | EGA

Sex Differences in Long COVID.

Not available

S-EPMC11755195 | biostudies-literature

Measurement of circulating viral antigens post-SARS-CoV-2 infection in a multicohort study.

Not available

S-EPMC11578795 | biostudies-literature

Differentiation of Prior SARS-CoV-2 Infection and Postacute Sequelae by Standard Clinical Laboratory Measurements in the RECOVER Cohort.

Not available

S-EPMC11408082 | biostudies-literature