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UnknownIdentifying potential therapeutic targets of Tang-Yi-Ping for the treatment of impaired glucose tolerance: a tandem mass tag-labeled quantitative proteomic analysis.
Not available
S-EPMC8576661 | biostudies-literature
UnknownMolecular characterization of China rabies virus vaccine strain.
Not available
S-EPMC3226571 | biostudies-literature
TranscriptomicsFunctional Genomic Analyses Identify Pathways Dysregulated by Progranulin Deficiency Implicating Wnt Signaling
Progranulin (GRN) mutations cause frontotemporal dementia (FTD), but GRN's function in the CNS remains largely unknown. To identify the pathways downstream of GRN, we used weighted genome co-expression network analysis (WGCNA) to develop a systems-level view of transcriptional alterations in a human...
ORGANISM(S): Homo sapiens 
2011-08-31 | E-GEOD-31772 | biostudies-arrayexpress
TranscriptomicsExpression data from Pin1 inhibitor ATRA and Pin1 knockdown
ATRA was identified as a Pin1 inhibitor via fluorescence polarization-based high throughput screening. We performed microarray expression profiling to demonstrate the similarity between ATRA and Pin1 KD at the genome-wide level APL NB4 cells in response to ATRRA or inducible Pin1 knockdown for 3 da...
ORGANISM(S): Homo sapiens 
2015-04-12 | E-GEOD-63059 | biostudies-arrayexpress
UnknownDiscovery of structural deletions in breast cancer predisposition genes using whole genome sequencing data from > 2000 women of African-ancestry.
Not available
S-EPMC9109261 | biostudies-literature
TranscriptomicsaCGH study of iPS cells from two normal subjects and two Parkinson's disease patients with parkin mutations
Parkinson's disease (PD) is defined by the degeneration of nigral dopaminergic (DA) neurons and can be caused by monogenic mutations of genes such as parkin. The lack of phenotype in parkin knockout mice suggests that human nigral DA neurons have unique vulnerabilities. Here we generate induced plur...
ORGANISM(S): Homo sapiens 
2012-01-21 | E-GEOD-35190 | biostudies-arrayexpress
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