Sort   by:  
 Page size 
UnknownAnti-endometriosis Mechanism of Jiawei Foshou San Based on Network Pharmacology.
Not available
S-EPMC6071511 | biostudies-literature
ProteomicsThe interactome of G3BP1 in UMUC3 cells
The HA-tagged G3BP1 complex was obtained by co-IP with anti-HA antibodies from UMUC3 cells stably expressing HA-tagged G3BP1. The purified immunoprecipitants were resolved on gradient SDS-PAGE, silver-stained, and subjected to MS analysis.
ORGANISM(S): Homo sapiens (Human) 
2024-05-23 | PXD035722 | Pride
Bladder cancer G3bp1 Umuc3 
UnknownJiawei Foshou San Induces Apoptosis in Ectopic Endometrium Based on Systems Pharmacology, Molecular Docking, and Experimental Evidence.
Not available
S-EPMC6855060 | biostudies-literature
UnknownTargeting PD-1/PD-L1 pathway in myelodysplastic syndromes and acute myeloid leukemia.
Not available
S-EPMC8889667 | biostudies-literature
UnknownEvaluation of clinical efficacy of integrated traditional Chinese and Western medicine in the treatment of acute respiratory distress syndrome.
Not available
S-EPMC7310857 | biostudies-literature
UnknownAssessing the genomic feature of Chinese patients with ampullary adenocarcinoma: potential therapeutic targets
Not available
S-EPMC10910796 | biostudies-literature
UnknownAnwulignan alleviates d-galactose induced renal damage by regulating Nrf2/ARE signaling pathway in mice.
Not available
S-EPMC8364582 | biostudies-literature
UnknownSoluble PD-L1 as a novel biomarker predicts poor outcomes and disease progression in de novo myelodysplastic syndromes.
Not available
S-EPMC11463091 | biostudies-literature
ProteomicsMS assay of proteins after CO-IP with GPNMB antibody in hippocampus area
Three mice were excised at day 7 after pilocarpine treatment to induce abundant GPNMB expression (Figures 1 and 2). Hippocampal proteins were extracted and pulled down by anti-GPNMB immunoprecipitation for mass-spectrometer assay.
ORGANISM(S): Mus Musculus 
PXD066376 |
ProteomicsZebrafish Study Provides Evidence for Porphyromonas gingivalis Outer Membrane Vesicles Eliciting Alzheimer's Disease-Like Pathologies
Periodontitis bacteria release vesicles that can harm the brain. In zebrafish, these vesicles caused brain injury, inflammation, movement problems, and Alzheimer’s-like changes, including amyloid plaques and enzyme activity. This study suggests periodontitis may contribute to Alzheimer’s disease.
ORGANISM(S): Danio Rerio 
PXD067862 |
Sort   by:  
 Page size