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Unknown
(29)
Proteomics
(1)
Organisms
Homo sapiens
(1)
Repository
pride
(1)
Tissue
Pancreas
(1)
Pancreatic ductal cell
(1)
Disease
Pancreatic ductal adenocarcinoma
(1)
Technology Type
Affinity purification coupled with mass spectrometry proteomics
(1)
Mass Spectrometry
(1)
Shotgun proteomics
(1)
Publication Date
2023
(1)
Release Date
2023
(4)
2022
(4)
2020
(4)
2024
(3)
2021
(3)
2017
(3)
2018
(2)
2016
(2)
2025
(1)
2019
(1)
2015
(1)
2009
(1)
Lab affiliation
Centre de Recherche en Cancérologie de Marseille, Inserm UMR1068 Parc Scientifique et Technologique de Luminy 163, Avenue de Luminy Case 915 13288 Marseille Cedex 9 FRANCE
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Growth/differentiation factor 15 controls ependymal and stem cell number in the V-SVZ.
Not available
S-EPMC10937156
|
biostudies-literature
Cite
The association between adverse events and outcome under checkpoint inhibitors: Where is the deal?
Not available
S-EPMC7708939
|
biostudies-literature
Cite
Pharmacokinetics and pharmacogenetics of Gemcitabine as a mainstay in adult and pediatric oncology: an EORTC-PAMM perspective.
Not available
S-EPMC4921117
|
biostudies-literature
Cite
Unraveling the complexity of therapeutic drug monitoring for monoclonal antibody therapies to individualize dose in oncology.
Not available
S-EPMC7981594
|
biostudies-literature
Cite
Like a Rolling Stone: Sting-Cgas Pathway and Cell-Free DNA as Biomarkers for Combinatorial Immunotherapy.
Not available
S-EPMC7464249
|
biostudies-literature
Cite
Clinical Pharmacokinetics and Pharmacodynamics of Immune Checkpoint Inhibitors.
Not available
S-EPMC6584248
|
biostudies-literature
Cite
Impact of renal impairment on dihydropyrimidine dehydrogenase (DPD) phenotyping.
Not available
S-EPMC10265596
|
biostudies-literature
Cite
Killing a fly with a sledgehammer: Atezolizumab exposure in real-world lung cancer patients.
Not available
S-EPMC10681534
|
biostudies-literature
Cite
Overcoming immuno-resistance by rescheduling anti-VEGF/cytotoxics/anti-PD-1 combination in lung cancer model.
Not available
S-EPMC10951825
|
biostudies-literature
Cite
Antiproliferative effect of ascorbic acid is associated with the inhibition of genes necessary to cell cycle progression.
Not available
S-EPMC2634969
|
biostudies-literature
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