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GenomicsHeparanase 2 Modulates Vascular Permeability via Heparan Sulfate-Dependent Growth Factor Signaling
Heparanase 2 Modulates Vascular Permeability via Heparan Sulfate-Dependent Growth Factor Signaling
PRJNA1154937 | ENA
GenomicsPhysiological Vascular Permeability Requires Induction of Endothelial NR4A1 by Progesterone Receptor [ChIP-Seq]
Vascular permeability is frequently associated with inflammation and it is triggered by chemokines and by a cohort of secreted permeability factors, such as VEGF. In contrast, here we showed that the physiological vascular permeability that precedes implantation is directly controlled by progestero...
ORGANISM(S): Homo sapiens 
2014-01-01 | E-GEOD-43786 | biostudies-arrayexpress
TranscriptomicsHeparanase 2 Modulates Vascular Permeability via Heparan Sulfate-Dependent Growth Factor Signaling
Heparan sulfate (HS) glycans attached to the apical surface of vascular endothelial cells (ECs) play an important role in regulating endothelial permeability and ligand recognition by cell-surface receptors. Shedding of HS from the EC surface increases vascular leakage and is associated with vascula...
ORGANISM(S): Danio rerio 
2025-07-15 | GSE276120 | GEO
UnknownSoluble CD40 ligand directly alters glomerular permeability and may act as a circulating permeability factor in FSGS.
Not available
S-EPMC5695800 | biostudies-literature
UnknownHeparanase 2 Modulates Vascular Permeability via Heparan Sulfate-Dependent Growth Factor Signaling.
Not available
S-EPMC12685291 | biostudies-literature
TranscriptomicsTranscription profiling of human neutrophils obtained after exposure endotoxin by bronchoscopic instillation reveals differential gene expression between air space and circulating neutrophils
Although the accumulation of neutrophils in the lungs and airways is common to many inflammatory lung diseases, including acute lung injury, the alterations that neutrophils undergo as they leave the peripheral circulation and migrate into the lungs have not been well characterized. Human volunteers...
ORGANISM(S): Homo sapiens 
2007-10-02 | E-GEOD-2322 | biostudies-arrayexpress
UnknownNovel mouse strains to study circulating permeability factor(s) in primary focal segmental glomerulosclerosis.
Not available
S-EPMC9499224 | biostudies-literature
GenomicsControl of VEGF-A trancriptional programs by pausing and genomic compartmentalization
Vascular endothelial growth factor A (VEGF-A) is a master regulator of vascular development and function. Consequently, VEGF-A regulated gene expression programs have been under heavy research. In this study we study the transcriptional regulation of VEGF-A-responsive genes in primary aortic endothe...
ORGANISM(S): Homo sapiens 
2014-10-27 | E-GEOD-52642 | biostudies-arrayexpress
TranscriptomicsExpression data from human healthy CD133+ bone marrow EPCs
Systemic lupus erythematosus (SLE) is characterized by increased vascular risk due to premature atherosclerosis independent of traditional risk factors. We previously proposed that interferon-α plays a crucial role in premature vascular damage in SLE. IFN-α alters the balance between endothelial cel...
ORGANISM(S): Homo sapiens 
2011-04-13 | E-GEOD-26951 | biostudies-arrayexpress
TranscriptomicsExpression data from human healthy EPCs/CACs
Systemic lupus erythematosus (SLE) is characterized by increased vascular risk due to premature atherosclerosis independent of traditional risk factors. We previously proposed that interferon-? plays a crucial role in premature vascular damage in SLE. IFN-? alters the balance between endothelial cel...
ORGANISM(S): Homo sapiens 
2011-04-13 | E-GEOD-26949 | biostudies-arrayexpress
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