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TranscriptomicsTranscription profiling of MIII celiac disease patients vs controls (M0) individuals
Comparison of MIII celiac disease patients to controls (M0) individuals.
ORGANISM(S): Homo sapiens 
2005-01-01 | E-MEXP-42 | biostudies-arrayexpress
UnknownCD38 expression on gluten-specific T cells is a robust marker of gluten re-exposure in coeliac disease.
Not available
S-EPMC6894002 | biostudies-literature
UnknownSelf-Reported Fractures in Dermatitis Herpetiformis Compared to Coeliac Disease.
Not available
S-EPMC5872769 | biostudies-literature
MetabolomicsPersistent Alterations in Plasma Lipid Profiles Before Introduction of Gluten in the Diet Associated With Progression to Celiac Disease

OBJECTIVES: Celiac disease (CD) is a chronic enteropathy characterized by an autoimmune reaction in the small intestine of genetically susceptible individuals. The underlying causes of autoimmune reaction and its effect on host metabolism remain largely unknown. Herein, we apply ...

2020-01-22 | MTBLS729 | MetaboLights
GenomicsGenome-wide methylation profiles in coeliac disease and healthy controls
Genome-wide methylation profiles in coeliac disease and healthy controls
PRJNA488062 | ENA
ProteomicsDifferent Binding Motifs of the Celiac Disease Associated HLA Molecules DQ2.5, DQ2.2 and DQ7.5 Revealed by Relative Quantitative Proteomics of Endogenous Peptide Repertoires
In this study relative quantitative analysis of endogenous peptides by mass spectrometry combined with neural network analysis have been used to address why the alpha- or beta-chain sharing human leukocyte antigen (HLA)-DQ molecules DQ2.5, DQ2.2 and DQ7.5 display different risks for celiac disease. ...
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2017-04-01 | MSV000080859 | MassIVE
UnknownFunctional implications of disease-specific variants in loci jointly associated with coeliac disease and rheumatoid arthritis.
Not available
S-EPMC4690494 | biostudies-literature
UnknownExperiences with Gluten-Free Bread: A Qualitative Study Amongst People with Coeliac Disease Participating in a Randomised Controlled Trial.
Not available
S-EPMC10706794 | biostudies-literature
TranscriptomicsArray comparative genomic hybridization in sporadic and coeliac disease-related small bowel adenocarcinomas
Background: The molecular pathogenesis of small intestinal adenocarcinomas (SBA) is not well understood. Defining its molecular pathogenesis may lead us to better clinical interventions. Aim: to identify the molecular changes characteristic of SBA. Methods: Forty-eight SBA (thirty-three non coelia...
ORGANISM(S): Homo sapiens 
2009-10-07 | E-GEOD-15470 | biostudies-arrayexpress
UnknownOutcome measures in coeliac disease trials: the Tampere recommendations.
Not available
S-EPMC6204961 | biostudies-literature
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