Sort   by:  
 Page size 
TranscriptomicsRegulation of neuronal gene and miRNA expression by the complement protein C1q associated with neuroprotection
Identification of transcriptional profiles stimulated by the complement protein C1q in rat immature neurons associated with the C1q-dependent neuroprotection observed in vitro. Use of the unbiased whole genome microarray approach to identify genes regulated by C1q. Immature neurons grown in vitro fo...
ORGANISM(S): Rattus norvegicus 
2011-03-07 | E-GEOD-18860 | biostudies-arrayexpress
ProteomicsComplement activation by AAV-neutralizing antibody complexes
IP-MS analysis of AAV9 empty capsid protein-protein interactions with serum from healthy human donors.
ORGANISM(S): Homo Sapiens (ncbitaxon:9606) 
2023-03-09 | MSV000091454 | MassIVE
TranscriptomicsModulation of gene expression by complement protein C1q in amyloid-beta injured neurons
Complement protein C1q is induced after injury in the brain and during Alzheimer's disease and has been shown to protect against amyloid-beta induced neuronal death. In this study, we used microarray approach to identify the pathways modulated by C1q that are associated with neuroprotection. Immatur...
ORGANISM(S): Rattus norvegicus 
2012-12-21 | E-GEOD-28886 | biostudies-arrayexpress
ProteomicsComplement deposition on Chlamydia trachomatis
Introduction Chlamydia trachomatis (C. trachomatis) is a Gram-negative bacterium and a common human pathogen. The World Health Organization (WHO) estimates that over 130 million people are infected with C. trachomatis globally each year and with increasing incidence. C. trachomatis causes long-lasti...
ORGANISM(S): Chlamydia trachomatis L2/434/Bu(f) Chlamydia trachomatis Homo sapiens (Human) Chlamydia trachomatis serovar L2 (strain 434/Bu / ATCC VR-902B) Chlamydia trachomatis L2/434/Bu(i) 
2020-11-23 | PXD019393 | Pride
C3 Complement Human Plasma Chlamydia trachomatis Lc-ms/ms 
TranscriptomicsDeciphering the genetic basis of Moraxella catarrhalis complement resistance: Gene expression analysis of Moraxella catarrhalis BBH18 upon exposure to normal human serum.
The complement system is an important part of the innate defense against invading pathogens. The ability to resist complement-mediated killing is considered to be an important virulence trait for the human-restricted respiratory tract pathogen M. catarrhalis, as most disease-associated M. catarrhali...
ORGANISM(S): Moraxella catarrhalis BBH18 
2014-06-23 | E-GEOD-52552 | biostudies-arrayexpress
GenomicsStress resilience is established during development and is regulated by complement factors
Stress resilience is established during development and is regulated by complement factors
PRJNA796216 | ENA
UnknownCompetition between antagonistic complement factors for a single protein on N. meningitidis rules disease susceptibility.
Not available
S-EPMC4273445 | biostudies-literature
ProteomicsExosome proteomics reveals the deregulation of coagulation, complement and lipid metabolism proteins in gestational diabetes mellitus
Exosomes are the smallest extracellular vesicles which are released during pregnancy by the placenta, umbilical cord, amniotic fluid and various cell membranes in the extracellular space. The content of exosomes during pregnancy is strongly related to various conditions such as gestational diabetes ...
ORGANISM(S): Homo sapiens (Human) 
2022-09-22 | PXD035673 | Pride
Proteomics Lipid metabolism Mass spectrometry Prothrombotic factors Complement system Gestational diabetes mellitus Platelet activation 
TranscriptomicsLocalized complement activation in the development of protective immunity against Ostertagia ostertagi infections in cattle
The gastrointestinal nematode Ostertagia ostertagi is one of major causal agents that contribute to production inefficiency in cattle industry in the temperate region of the world. One of pathophysiological factors that lead to reduced weight gain and milk yield is altered gastrointestinal functions...
ORGANISM(S): Bos taurus 
2010-05-05 | E-GEOD-20927 | biostudies-arrayexpress
TranscriptomicsRetinal Pigment Epithelial Cells Upregulate Expression of Complement Factors after Co-culture with Activated T Cells
In this study we examined the effect of T cell-derived cytokines on retinal pigment epithelial (RPE) cells with respect to expression of complement components. We used an in vitro co-culture system in which CD3/CD28-activated human T cells were separated from the human RPE cell line (ARPE-19) by a m...
ORGANISM(S): Homo sapiens 
2011-02-02 | GSE17938 | GEO
Sort   by:  
 Page size