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Oxidant-resistant LRRC8A/C anion channels support superoxide production by NADPH oxidase 1.
Not available
S-EPMC8600960
|
biostudies-literature
Cite
Depletion of Mitochondrial Cyclophilin D in Endothelial and Smooth Muscle Cells Attenuates Vascular Dysfunction and Hypertension.
Not available
S-EPMC11931617
|
biostudies-literature
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Breast cancer chemotherapy induces vascular dysfunction and hypertension through a NOX4-dependent mechanism.
Not available
S-EPMC9246378
|
biostudies-literature
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Poldip2 knockdown inhibits vascular smooth muscle proliferation and neointima formation by regulating the expression of PCNA and p21.
Not available
S-EPMC6393166
|
biostudies-literature
Cite
LRRC8A channels support TNFα-induced superoxide production by Nox1 which is required for receptor endocytosis.
Not available
S-EPMC5206799
|
biostudies-literature
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Coupling of phagocytic NADPH oxidase activity and mitochondrial superoxide production.
Not available
S-EPMC9366351
|
biostudies-literature
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NADPH oxidase 4 mediates TGF-β-induced smooth muscle α-actin via p38MAPK and serum response factor.
Not available
S-EPMC3032946
|
biostudies-literature
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Nox2-induced production of mitochondrial superoxide in angiotensin II-mediated endothelial oxidative stress and hypertension.
Not available
S-EPMC3887459
|
biostudies-literature
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Mitochondrial CypD Acetylation Promotes Endothelial Dysfunction and Hypertension.
Not available
S-EPMC11116043
|
biostudies-literature
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Mitochondrial Deacetylase Sirt3 Reduces Vascular Dysfunction and Hypertension While Sirt3 Depletion in Essential Hypertension Is Linked to Vascular Inflammation and Oxidative Stress.
Not available
S-EPMC7035170
|
biostudies-literature
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