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TranscriptomicsMolecular profiles (HG-U95A) of dystrophin-deficient and normal human muscle
Molecular profiles of dystophin-deficient patients and normal human skeletal muscles on Affymetrix HG-U95A arrays Keywords = DMD Keywords = Duchenne muscular dystrophy Keywords = dystrophin Keywords = Affymetrix U95A array Keywords = skeletal muscle Keywords = gene expression profiles Keywords: othe...
ORGANISM(S): Homo sapiens 
2004-01-29 | GSE1004 | GEO
TranscriptomicsMolecular profiles (HG-U95A) of dystrophin-deficient and normal human muscle
Molecular profiles of dystophin-deficient patients and normal human skeletal muscles on Affymetrix HG-U95A arrays Keywords = DMD Keywords = Duchenne muscular dystrophy Keywords = dystrophin Keywords = Affymetrix U95A array Keywords = skeletal muscle Keywords = gene expression profiles Keywords: othe...
ORGANISM(S): Homo sapiens 
2004-01-29 | E-GEOD-1004 | biostudies-arrayexpress
GenomicsHomo sapiens
Molecular profiles (HG-U95A) of dystrophin-deficient and normal human muscle
PRJNA87057 | ENA
TranscriptomicsIPSC derived cardiac fibroblasts of DMD patients show compromised actin microfilaments, metabolic shift and pro-fibrotic phenotype.
Duchenne muscular dystrophy (DMD) is a severe form of muscular dystrophy caused by mutations in the dystrophin gene. We characterized which isoforms of dystrophin were expressed by human induced pluripotent stem cell (hiPSC)-derived cardiac fibroblasts obtained from control and DMD patients. Distinc...
ORGANISM(S): Homo sapiens 
2023-09-30 | GSE237014 | GEO
ProteomicsRescued ontogenesis in human DMD mini-muscles driven by tricyclo-DNA exon skipping-mediated dystrophin restoration
Duchenne muscular dystrophy (DMD) is a lethal X-linked recessive disorder caused by loss of function mutations in the dystrophin gene. There is currently no cure for DMD although various promising approaches are progressing through human clinical trials. Among them, exon-skipping therapy using antis...
ORGANISM(S): Homo sapiens (Human) 
2026-09-14 | PXD060394 | Pride
Dystophin 3d-cell culture Dmd 
GenomicsIPSC derived cardiac fibroblasts of DMD patients show compromised actin microfilaments, metabolic shift and pro-fibrotic phenotype.
IPSC derived cardiac fibroblasts of DMD patients show compromised actin microfilaments, metabolic shift and pro-fibrotic phenotype.
PRJNA993401 | ENA
GenomicsRNA sequencing of TA skeletal muscle of mice with conditional ablation of dystrophin within the myofiber
RNA sequencing of TA skeletal muscle of mice with conditional ablation of dystrophin within the myofiber
PRJNA1200112 | ENA
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