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ProteomicsE-selectin Affinity Glycoproteomics Reveals Neuroendocrine Proteins and the Secretin Receptor as a Poor Prognosis Signature in Colorectal Cancer
Colorectal cancer (CRC) cells express glycoproteins with sialylated Lewis antigens (sLe), crucial for metastasis via E-selectin binding. However, these glycoepitopes lack cancer specificity, and E-selectin-targeted glycoproteins are unknown. Here we established a framework for identifying metastasi...
ORGANISM(S): Homo sapiens (Human) 
2024-11-11 | PXD054727 | Pride
Cancer glycoproteome Colorectal cancer E-selectin Secretin receptor Metastasis 
TranscriptomicsExpression data from macrophages of E-selectin+/+ and E-selectin-/- mouse infected with Listeria monocytogenes in vivo on day3
We used microarrays to detail the global gene expression in peritoneal macrophages (PM) from E-selectin+/+ and E-selectin-/- mouse infected with Listeria Monocytogenes in vivo on day3
ORGANISM(S): Mus musculus 
2018-01-25 | GSE90808 | GEO
GenomicsCA19-9 promotes liver metastasis of pancreatic cancer through E-selectin mediated extravasation
CA19-9 promotes liver metastasis of pancreatic cancer through E-selectin mediated extravasation
PRJNA1503584 | ENA
ProteomicsE-selectin-mediated rapid NLRP3 inflammasome activation regulates S100A8/A9 release from neutrophils via transient gasdermin D pore formation
S100A8/A9 is a proinflammatory mediator released by myeloid cells during many acute and chronic inflammatory disorders. However, the precise mechanism of its release from the cytosolic compartment of neutrophils is still elusive. We report here that E-selectin-induced rapid S100A8/A9 release during ...
ORGANISM(S): Homo sapiens (Human) 
2023-10-25 | PXD041652 | Pride
Neutrophile Inflammation Gasdermin d Transient pore formation 
GenomicsE-Selectin/AAV2/2 Gene Therapy Alters Angiogenesis and Inflammatory Gene Profiles in Mouse Gangrene Model [Inflammation]
E-Selectin/AAV2/2 Gene Therapy Alters Angiogenesis and Inflammatory Gene Profiles in Mouse Gangrene Model [Inflammation]
PRJNA831894 | ENA
GenomicsE-Selectin/AAV2/2 Gene Therapy Alters Angiogenesis and Inflammatory Gene Profiles in Mouse Gangrene Model [Angiogenesis]
E-Selectin/AAV2/2 Gene Therapy Alters Angiogenesis and Inflammatory Gene Profiles in Mouse Gangrene Model [Angiogenesis]
PRJNA831895 | ENA
TranscriptomicsE-Selectin/AAV2/2 Gene Therapy Alters Angiogenesis and Inflammatory Gene Profiles in Mouse Gangrene Model [Inflammation]
For patients with chronic limb-threatening ischemia and limited revascularization options, alternate means for therapeutic angiogenesis and limb salvage are needed. E-selectin is a cell adhesion molecule that is critical for inflammation and neovascularization in areas of wound healing and ischemia....
ORGANISM(S): Mus musculus 
2022-04-29 | GSE201479 | GEO
TranscriptomicsE-Selectin/AAV2/2 Gene Therapy Alters Angiogenesis and Inflammatory Gene Profiles in Mouse Gangrene Model [Angiogenesis]
For patients with chronic limb-threatening ischemia and limited revascularization options, alternate means for therapeutic angiogenesis and limb salvage are needed. E-selectin is a cell adhesion molecule that is critical for inflammation and neovascularization in areas of wound healing and ischemia....
ORGANISM(S): Mus musculus 
2022-04-29 | GSE201476 | GEO
GenomicsMus musculus
Expression data from macrophages of E-selectin+/+ and E-selectin-/- mouse infected with Listeria monocytogenes in vivo on day3
PRJNA355996 | ENA
TranscriptomicsCA19-9 promotes liver metastasis of pancreatic cancer through E-selectin mediated extravasation
Pancreatic ductal adenocarcinoma (PDAC) frequently metastasizes to the liver, which drives patient mortality. CA19-9 is elevated in most PDAC tumors and is widely used as a clinical biomarker. Elevated serum levels are associated with poor outcomes. However, whether CA19-9 functionally contributes t...
ORGANISM(S): Mus musculus 
2026-09-04 | GSE341736 | GEO
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