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GenomicsRNA-sequencing explores the transcriptional profile of Wild Type and Fabp3-null mice under physiologicl and cardiac hypertrophic conditions
RNA-sequencing explores the transcriptional profile of Wild Type and Fabp3-null mice under physiologicl and cardiac hypertrophic conditions
PRJNA736943 | ENA
TranscriptomicsRNA-sequencing explores the transcriptional profile of Wild Type and Fabp3-null mice under physiologicl and cardiac hypertrophic conditions
Background: Cardiac hypertrophy was accompanied by various cardiovascular diseases (CVDs), due to the high global incidence and mortality of CVDs, it has become increasingly critical to characterize the pathogenesis of cardiac hypertrophy. We aimed to determine the metabolic effects of fatty acid bi...
ORGANISM(S): Mus musculus 
2021-06-12 | GSE177041 | GEO
UnknownLight up heart-type fatty acid binding protein (FABP3) with a novel fluorine-18 labelled selective FABP3 ligand.
Not available
S-EPMC11564661 | biostudies-literature
UnknownFABP3 Deficiency Exacerbates Metabolic Derangement in Cardiac Hypertrophy and Heart Failure via PPARα Pathway.
Not available
S-EPMC8387950 | biostudies-literature
UnknownE2F8 promotes hepatic steatosis through FABP3 expression in diet-induced obesity in zebrafish.
Not available
S-EPMC4456805 | biostudies-literature
UnknownFABP3-mediated membrane lipid saturation alters fluidity and induces ER stress in skeletal muscle with aging.
Not available
S-EPMC7653047 | biostudies-literature
UnknownTumor-Targeting Peptides: The Functional Screen of Glioblastoma Homing Peptides to the Target Protein FABP3 (MDGI).
Not available
S-EPMC7409020 | biostudies-literature
UnknownThe oxidized phospholipid PGPC impairs endothelial function by promoting endothelial cell ferroptosis via FABP3.
Not available
S-EPMC10864338 | biostudies-literature
UnknownSilencing the FABP3 gene in insulin-secreting cells reduces fatty acid uptake and protects against lipotoxicity.
Not available
S-EPMC11628584 | biostudies-literature
UnknownOverexpression of FABP3 inhibits human bone marrow derived mesenchymal stem cell proliferation but enhances their survival in hypoxia.
Not available
S-EPMC4049325 | biostudies-literature
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