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ProteomicsProteomic analysis of Fam83h deficient and wild type ileum
We generated Fam83h-deficient mice (Fam83hem2(IMPC)Ccpcz, Fam83h-/-) and mice lacking a part of N-terminal CK1-binding domain (Fam83h∆87/∆87). Co-IP with anti-FAM83H antibody has confirmed the FAM83H–CK1 interaction in Fam83hwt/wt samples, which was absent in Fam83h-/- lysates. Additionally, samples...
ORGANISM(S): Mus musculus (Mouse) 
2025-09-08 | PXD064193 | Pride
Fam83h-deficient mice Fam83h 
GenomicsFAM83H-AS1 silencing in MCF7 breast cancer cells
FAM83H-AS1 silencing in MCF7 breast cancer cells
PRJNA558978 | ENA
GenomicsEffect of FAM83H-AS1 knockdown in MDA-MB-468 cells [cellseq]
Effect of FAM83H-AS1 knockdown in MDA-MB-468 cells [cellseq]
PRJNA1225303 | ENA
GenomicsRNA-sequencing reveals signaling pathway associated with cell adhesion in Fam83h-mutated cells
RNA-sequencing reveals signaling pathway associated with cell adhesion in Fam83h-mutated cells
PRJNA801618 | ENA
TranscriptomicsFAM83H-AS1 silencing in MCF7 breast cancer cells
We silenced FAM83H-AS1 shRNAs in cell line MCF7 carried a ~75% silencing compared to thenegative control (NC). We evaluated the role of FAM83H-AS1 on oncogenic phenotypes in the MCF7 breast cancer cell line model. The knockdown of FAM83H-AS1 was achieved with ~75% of silencing efficiency. A complete...
ORGANISM(S): Homo sapiens 
2020-12-12 | GSE135466 | GEO
GenomicsFAM83H-AS1 is a non-coding oncogenic driver and therapeutic target of lung adenocarcinoma
FAM83H-AS1 is a non-coding oncogenic driver and therapeutic target of lung adenocarcinoma
PRJNA669700 | ENA
TranscriptomicsRNA-sequencing reveals signaling pathway associated with cell adhesion in Fam83h-mutated cells
Truncation mutations in family with sequence similarity, member H (FAM83H) gene cause autosomal dominant hypocalcified amelogenesis imperfecta (ADHCAI). The aim of this study was to explore the effects of truncated FAM83H on enamel development. High throughput RNA-sequencing was used to detect the d...
ORGANISM(S): Mus musculus 
2024-01-28 | GSE195645 | GEO
TranscriptomicsEffect of FAM83H-AS1 knockdown in MDA-MB-468 cells [cellseq]
To investigate the role of lncRNA FAM83H-AS1 in breast cancer, we used shRNA to silence FAM83H-AS1 in MDA-MB-468 cells and performed RNA-seq analysis.
ORGANISM(S): Homo sapiens 
2026-02-01 | GSE289922 | GEO
TranscriptomicsFAM83H-AS1 is a non-coding oncogenic driver and therapeutic target of lung adenocarcinoma
Our study suggested that FAM83H-AS1 was a potential oncogenic driver due to chromosome 8q24 amplification in lung adenocarcinoma. To investigate the molecular mechanism of FAM83H-AS1, we performed high-thoughput RNA sequencing (RNA-Seq) assays after the silence of FAM83H-AS1 in A549 cell lines.
ORGANISM(S): Homo sapiens 
2020-10-20 | GSE159559 | GEO
UnknownLncRNA FAM83H-AS1 promotes the malignant progression of pancreatic ductal adenocarcinoma by stabilizing FAM83H mRNA to protect β-catenin from degradation
Not available
S-EPMC9520839 | biostudies-literature
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