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TranscriptomicsMolecular characterization of chronic antibody mediated rejection in kidney transplantation (microRNA)
Chronic antibody-mediated rejection (CAMR) represents the main cause of kidney graft loss, but its pathogenesis is unclear. In order to uncover the molecular mechanisms underlying this condition, we characterized the molecular signature of circulating peripheral blood mononuclear cells and, separate...
ORGANISM(S): Homo sapiens 
2015-12-10 | E-GEOD-51676 | biostudies-arrayexpress
TranscriptomicsMolecular characterization of chronic antibody mediated rejection in kidney transplantation (mRNA)
Chronic antibody-mediated rejection (CAMR) represents the main cause of kidney graft loss, but its pathogenesis is unclear. In order to uncover the molecular mechanisms underlying this condition, we characterized the molecular signature of circulating peripheral blood mononuclear cells and, separate...
ORGANISM(S): Homo sapiens 
2015-12-10 | E-GEOD-51675 | biostudies-arrayexpress
TranscriptomicsMolecular characterization of chronic antibody mediated rejection in kidney transplantation (mRNA CD4)
Chronic antibody-mediated rejection (CAMR) represents the main cause of kidney graft loss, but its pathogenesis is unclear. In order to uncover the molecular mechanisms underlying this condition, we characterized the molecular signature of circulating peripheral blood mononuclear cells and, separate...
ORGANISM(S): Homo sapiens 
2015-12-10 | E-GEOD-64261 | biostudies-arrayexpress
TranscriptomicsGenome-wide analysis of differentially expressed genes and splicing isoforms in clear cell Renal Cell Carcinoma
In this study we performed a genome wide analysis of the entire complement of mRNAs in clear cell renal cell carcinomas (ccRCC) by means of the Affymetrix Exon Array platform. The analyses were performed both at gene and exon level. Under our parameters over 2,000 genes resulted differentially expre...
ORGANISM(S): Homo sapiens 
2013-09-06 | E-GEOD-47032 | biostudies-arrayexpress
UnknownClinical features and long-term outcome of nephrotic syndrome associated with heterozygous NPHS1 and NPHS2 mutations.

Background and objectives

Mutations in nephrin (NPHS1) and podocin (NPHS2) genes represent a major cause of idiopathic nephrotic syndrome (NS) in children. It is not yet clear whether the presence of a single mutation acts as a modifier of the clinical course of NS.

Design, setting, parti...

S-EPMC2689885 | biostudies-literature
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