H3k27 methylation

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1. Cancer Signalling Research Group, School of Biomedical Sciences and Pharmacy, College of Health, Medicine and Wellbeing, University of Newcastle, Callaghan, NSW, AUSTRALIA 2. Precision Medicine Research Program, Hunter Medical Research Institute, New Lambton Heights, NSW, AUSTRALIA 3. Paediatric Program, Mark Hughes Foundation Centre for Brain Cancer Research, College of Health, Medicine, and Wellbeing, Callaghan, NSW, AUSTRALIA (1)
1. Novo Nordisk Foundation Center for Protein Research, University of Copenhagen, Copenhagen, Denmark; 2. Proteomics and Singal Transduction, Max Planck Institute of Biochemistry, Martinsried, Germany (1)
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Key Laboratory of Biomedical Information Engineering of Ministry of Education, School of Life Science and Technology, Xi'an Jiaotong University, Xi'an 710049, Shaanxi Province, China. (1)
Laboratoire de Spectrometrie de Masse Proteomique Institut Curie PSL Research University (1)
Max-Planck Institute of Biochemistry, Laboratory of Chromatin Biology, Martinsried, Germany (1)
Novo Nordisk Foundation Center for Protein Research (CPR), University of Copenhagen, Copenhagen, Denmark. (1)
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School of Biosciences, University of Sheffield. (1)
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Université Paris Cité, CNRS, Institut Jacques Monod, 15 rue Hélène Brion, F-75013 Paris, France (1)
Vascular Dysfunction Department European Center for Angioscience Medical Faculty Mannheim Heidelberg University (1)

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Loss of H3K27 monomethylation leads to DNA-methylation-independent release of gene silencing

Constitutive heterochromatin in Arabidopsis thaliana is marked by repressive chromatin modifications including DNA methylation, histone 3 dimethylation at lysine 9 (H3K9me2), and monomethylation at lysine 27 (H3K27me1). The enzymes catalyzing DNA methylation and H3K9me2 have been identified and muta...

ORGANISM(S): Arabidopsis thaliana

2009-05-31 | E-GEOD-16071 | biostudies-arrayexpress

Replicative senescence of mesenchymal stem cells causes DNA methylation changes which correlate with repressive histone marks

Cells in culture undergo replicative senescence. In this study, we analyzed functional, genetic and epigenetic sequels of long‐term culture in human mesenchymal stem cells (MSC). Already within early passages the fibroblastoid colony‐ forming unit (CFU‐f) frequency and the differentiation potential ...

ORGANISM(S): Homo sapiens

2011-10-27 | E-GEOD-26519 | biostudies-arrayexpress

H3K36 methylation antagonizes PRC2-mediated H3K27 methylation.

Not available

S-EPMC3048685 | biostudies-literature

Histone H3K27 methylation states are sequentially catalyzed in cycling cells

PRJNA1452821 | ENA

Gene expression and H3K27 tri-methylation analysis of meristematic tissue (Me) and young leaves (Le) isolated by manual dissection of clv3-9 plants

This SuperSeries is composed of the following subset Series: GSE24464: Expression analysis of meristematic tissue (Me) and young leaves (Le) isolated by manual dissection of clv3-9 plants GSE24474: H3K27 tri-methylation analysis of meristematic tissue (Me) and young leaves (Le) isolated by manual di...

ORGANISM(S): Arabidopsis thaliana

2011-04-20 | E-GEOD-24507 | biostudies-arrayexpress

The histone H3.3K27M mutation in pediatric glioma reprograms H3K27 methylation and gene expression

Recent studies have identified a Lys 27-to-methionine (K27M) mutation at one allele of H3F3A, one of the two genes encoding histone H3 variant H3.3, in 60% of high-grade pediatric glioma cases. The median survival of this group of patients after diagnosis is ∼1 yr. Here we show that the levels of H3...

ORGANISM(S): Homo sapiens

2014-09-20 | E-GEOD-61586 | biostudies-arrayexpress

Orchestration of H3K27 methylation: mechanisms and therapeutic implication.

Not available

S-EPMC5756243 | biostudies-literature

G9a orchestrates PCL3 and KDM7A to promote histone H3K27 methylation.

Not available

S-EPMC4685317 | biostudies-literature

Accurate H3K27 methylation can be established de novo by SUZ12-directed PRC2.

Not available

S-EPMC5842896 | biostudies-literature

Molecular Mechanisms Directing PRC2 Recruitment and H3K27 Methylation.