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GenomicsThe alteration of 5-hydroxymethylcytosine modification contributes to ischemic brain injury [5hmC-seq]
Epigenetic modifications, such as cytosine methylation and histone modification, have been shown involved in the pathology of ischemic brain injury. Recent works have implicated 5-hydroxymethylcytosine (5hmC), a DNA base derived from 5-methylcytosine (5mC) through the oxidation by Ten-Eleven Translo...
ORGANISM(S): Mus musculus 
2015-09-08 | E-GEOD-67188 | biostudies-arrayexpress
UnknownSpecies delimitation of Margattea cockroaches from China, with seven new species (Blattodea, Ectobiidae, Pseudophyllodromiinae).
Not available
S-EPMC8128847 | biostudies-literature
UnknownMutation of Candida tropicalis by irradiation with a He-Ne laser to increase its ability to degrade phenol.
Not available
S-EPMC1797104 | biostudies-literature
UnknownChemopreventive effects of Ku-jin tea against AOM-induced precancerous colorectal lesions in rats and metabolomic analysis.
Not available
S-EPMC5698479 | biostudies-literature
UnknownStudy on the mechanism of Jin Gui Shen Qi Pill in the treatment of erectile dysfunction based on bioinformatics analysis.
Not available
S-EPMC9678517 | biostudies-literature
UnknownEfficacy and safety of Jin-shui Huan-xian granule for idiopathic pulmonary fibrosis: study protocol for a multicenter, randomized, double-blind, placebo-controlled trial.
Not available
S-EPMC9438282 | biostudies-literature
TranscriptomicsCombined loss of Tet1 and Tet2 promotes B-cell, but not myeloid malignancies in mice
TET1/2/3 are methylcytosine dioxygenases regulating cytosine hydroxymethylation in the genome. Tet1 and Tet2 are abundantly expressed in HSC/HPCs and implicated in the pathogenesis of hematological malignancies. Tet2-deletion in mice causes myeloid malignancies, while Tet1-null mice develop B-cell l...
ORGANISM(S): Mus musculus 
2015-11-12 | E-GEOD-73611 | biostudies-arrayexpress
ProteomicsER-phagy restrains inflammatory responses through receptor UBAC2
ER-phagy, a selective degradation of endoplasmic reticulum (ER) fragment, plays a critical role in maintaining the ER homeostasis. Dysregulation of ER-phagy is involved in the unfolded protein response (UPR), which serves as a vital clue to evoke inflammatory disease. However, the molecular mechanis...
ORGANISM(S): Homo sapiens (Human) 
2025-05-07 | PXD052877 | Pride
Er-phagy  ubac2  mark2  inflammatory responses  colitis 
ProteomicsER-phagy restrains inflammatory responses through receptor UBAC2
ER-phagy, a selective degradation of endoplasmic reticulum (ER) fragment, plays a critical role in maintaining the ER homeostasis. Dysregulation of ER-phagy is involved in the unfolded protein response (UPR), which serves as a vital clue to evoke inflammatory disease. However, the molecular mechanis...
ORGANISM(S): Homo sapiens (Human) 
2025-05-07 | PXD052418 | Pride
Er-phagy  ubac2  mark2  inflammatory responses  colitis 
MetabolomicsDisorders of gut microbiota and fecal-serum metabolic patterns are associated with pulmonary tuberculosis and pulmonary tuberculosis co-morbid type 2 diabetes mellitus

BACKGROUND: Pulmonary tuberculosis (PTB) and diabetes mellitus (DM) are prevalent chronic diseases with substantial implications for human health. DM patients are more susceptible to PTB, which exacerbates diabetes-related complications. However, the complex molecular mechanisms ...

2023-12-15 | MTBLS8729 | MetaboLights
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