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TranscriptomicsCcpA, a Pleiotropic Key Regulator in Butanol-producing Clostridium acetobutylicum ATCC 824
Clostridium acetobutylicum is a typical bacterium of major importance to industrial butanol production. In order to dissect the regulatory network pertaining to the industrial application of this bacterium, catabolite control protein A (CcpA) was investigated for its global function by DNA microarra...
ORGANISM(S): Clostridium acetobutylicum 
2012-08-30 | E-GEOD-33364 | biostudies-arrayexpress
UnknownInhibition of cystathionine β-synthase promotes apoptosis and reduces cell proliferation in chronic myeloid leukemia.
Not available
S-EPMC7870845 | biostudies-literature
UnknownLandscape of enhancer disruption and functional screen in melanoma cells.
Not available
S-EPMC10614365 | biostudies-literature
TranscriptomicsComparative genomic and transcriptomic analysis revealed genetic characteristics related to solvent formation and xylose utilization in Clostridium acetobutylicum EA 2018
Transcriptomic profiling of gene expression in EA 2018 relative to that of ATCC824 revealed several key genes related to solvent formation. For example, spo0A and adhEII have higher expression level, and most of the acid formation related genes have lower expression level in EA 2018. Cells for RNA ...
ORGANISM(S): Clostridium acetobutylicum EA 2018 
2012-07-22 | E-GEOD-23071 | biostudies-arrayexpress
UnknownThe moderated-mediation role of risk perception and intolerance of uncertainty in the association between residual symptoms and psychological distress: a cross-sectional study after COVID-19 policy lifted in China.
Not available
S-EPMC10874086 | biostudies-literature
UnknownAn autoimmune pleiotropic SNP modulates IRF5 alternative promoter usage through ZBTB3-mediated chromatin looping.
Not available
S-EPMC9984425 | biostudies-literature
UnknownDiphthamide deficiency promotes association of eEF2 with p53 to induce p21 expression and neural crest defects.
Not available
S-EPMC11053169 | biostudies-literature
ProteomicsMuscular lactate modulates phase transition-driven lactate body secretion to induce liver fibrosis during long-term overtraining
Exercise, as the fundamental therapy for metabolic diseases, often exhibits a dose-dependent fashion. However, the adverse effects of overtraining and underlying mechanisms remain unclear. Here, we show that long-term overtraining induces hepatic fibrosis in humans and mice. Mechanistically, excessi...
ORGANISM(S): Mus Musculus 
2024-05-21 | PXD052424 |
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