Sort   by:  
 Page size 
UnknownMutations in the ANGPTL3 gene and familial combined hypolipidemia: a clinical and biochemical characterization.
Not available
S-EPMC5393441 | biostudies-literature
UnknownExome sequencing, ANGPTL3 mutations, and familial combined hypolipidemia.
Not available
S-EPMC3008575 | biostudies-literature
UnknownMANF/EWSR1/ANXA6 pathway might as the bridge between hypolipidemia and major depressive disorder.
Not available
S-EPMC9803680 | biostudies-literature
GenomicsLipid restriction in Familial Hypolipidemia 2 amplifies type I interferon response and suppresses IL-1beta production in a prenylation-dependent fashion
Lipid restriction in Familial Hypolipidemia 2 amplifies type I interferon response and suppresses IL-1beta production in a prenylation-dependent fashion
PRJNA1321758 | ENA
UnknownAngiopoietin-like protein 3 (ANGPTL3) deficiency and familial combined hypolipidemia.
Not available
S-EPMC6477171 | biostudies-literature
UnknownLipid deprivation amplifies type I IFN responses in monocytes through prenylation: insights from familial combined hypolipidemia type 2.
Not available
S-EPMC12764162 | biostudies-literature
TranscriptomicsLipid restriction in Familial Hypolipidemia 2 amplifies type I interferon response and suppresses IL-1beta production in a prenylation-dependent fashion
Familial Combined Hypolipidemia (FHBL2) is a genetic disorder caused by loss-of-function mutations in the Angiopoietin-like 3 (ANGPTL3) gene. FHBL2 subjects exhibit hypolipidemia and protection from cardiometabolic diseases. Here, we explored the hypothesis that immunometabolic events contribute to ...
ORGANISM(S): Homo sapiens 
2025-11-28 | GSE307462 | GEO
UnknownClinical characteristics and plasma lipids in subjects with familial combined hypolipidemia: a pooled analysis.
Not available
S-EPMC3826694 | biostudies-literature
UnknownPPARalpha: energy combustion, hypolipidemia, inflammation and cancer.
Not available
S-EPMC2858266 | biostudies-literature
ProteomicsGut-liver axis calibrates intestinal stem cell fitness via PEDF
The gut and liver have been recognized to mutually communicate through the biliary tract, portal vein and systemic circulation, but it remains unclear how this gut-liver axis regulates intestinal physiology. Through hepatectomy, transcriptomics and proteomics profiling, we identified pigment epithel...
ORGANISM(S): Mus Musculus (ncbitaxon:10090) 
2023-03-03 | MSV000091409 | MassIVE
Sort   by:  
 Page size