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TranscriptomicsExpression data from human amnion mesenchymal cells treated with interleukin-1-beta for 1hr and 8hr
Premature birth continues to be a challenging pregnancy complication, and a body of literature indicates that inflammation can contribute to premature delivery by converting a receptive uterine environment to a hostile one. Cytokines have been demonstrated to provoke up-regulation of inflammatory g...
ORGANISM(S): Homo sapiens 
2011-06-02 | E-GEOD-26315 | biostudies-arrayexpress
TranscriptomicsTranscription profiling by array of rat ventricular cardiac myocytes treated with interleukin 1 beta
Study of the effects of interleukin 1 beta on transcript expression in ventricular cardiac myocytes
ORGANISM(S): Rattus norvegicus 
2010-04-01 | E-MEXP-1960 | biostudies-arrayexpress
GenomicsEvaluating the Role of Interleukin 1 Beta in Mesial Temporal Lobe Epilepsy by candidate SNPs
We aimed to investigate the role of interleukin-1 beta in the mechanisms underlying mesial temporal lobe epilepsy with hippocampal sclerosis (MTLE+HS). We assessed a cohort of patients with MTLE+HS and healthy controls. We investigated the genetic association with single nucleotide polymorphisms (SN...
ORGANISM(S): Homo Sapiens 
2021-04-17 | PRJEB44301 | EVA
TranscriptomicsThe role of interleukin-1 beta in the pathophysiology of Schnitzler’s syndrome
Schnitzler’s syndrome (SchS) is a disabling autoinflammatory disorder, characterized by chronic urticaria a chronic urticarial rash, an M-protein, arthralgia, and other signs of systemic inflammation. Anti-interleukin-1 (IL-1) beta antibodies are highly effective, but the pathophysiology is still la...
ORGANISM(S): Homo sapiens 
2015-08-01 | E-GEOD-70019 | biostudies-arrayexpress
TranscriptomicsExpression data from primary term human decidual cells treated with interleukin-1-beta for 6 hours.
Preterm birth is an important unsolved clinical problem. Despite advanced treatments, infants who survive prematurity remain at increased risk for permanent disabilities. In approximately one-third of cases, prematurity is related to infection and/or inflammation, which renders hostile the normall...
ORGANISM(S): Homo sapiens 
2016-07-03 | E-GEOD-58220 | biostudies-arrayexpress
GenomicsInhibition of interleukin-1 receptor-associated kinase (IRAK)-4 provides partial rescue of interleukin-1 beta induced functional and gene expression changes in equine tendon cells.
Inhibition of interleukin-1 receptor-associated kinase (IRAK)-4 provides partial rescue of interleukin-1 beta induced functional and gene expression changes in equine tendon cells.
PRJNA1279161 | ENA
GenomicsExogenous interleukin-1 beta stimulation regulates equine tenocyte function and gene expression which can only be rescued by pharmacological inhibition of interleukin 1 receptor, but not nuclear factor kappa B, signalling
Exogenous interleukin-1 beta stimulation regulates equine tenocyte function and gene expression which can only be rescued by pharmacological inhibition of interleukin 1 receptor, but not nuclear factor kappa B, signalling
PRJNA914045 | ENA
TranscriptomicsInhibition of interleukin-1 receptor-associated kinase (IRAK)-4 provides partial rescue of interleukin-1 beta induced functional and gene expression changes in equine tendon cells.
We investigated if the daily application of the interleukin-1 receptor-associated kinase (IRAK)-4 inhibitor PF-06650833 could rescue the effect of interleukin-1 beta on equine tendon cells cultured in 3D
ORGANISM(S): Equus caballus 
2025-11-06 | GSE300167 | GEO
GenomicsPancreatic β-cell interleukin-22 receptor subunit alpha 1 deficiency impairs β-cell function in type 2 diabetes via cytochrome b5 reductase 3 [BKO]
Pancreatic β-cell interleukin-22 receptor subunit alpha 1 deficiency impairs β-cell function in type 2 diabetes via cytochrome b5 reductase 3 [BKO]
PRJNA1185158 | ENA
GenomicsPancreatic β-cell interleukin-22 receptor subunit alpha 1 deficiency impairs β-cell function in type 2 diabetes via cytochrome b5 reductase 3 [shRNA]
Pancreatic β-cell interleukin-22 receptor subunit alpha 1 deficiency impairs β-cell function in type 2 diabetes via cytochrome b5 reductase 3 [shRNA]
PRJNA1185161 | ENA
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