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MetabolomicsUPLC-Q-TOF/MS-Based Plasma and Urine Metabolomics Contribute to the Diagnosis of Sepsis
In this study, we aimed to identify potential metabolic biomarkers that can improve the diagnostic accuracy of sepsis. Sixty-six patients including 30 septic and 36 nonsepsis patients from an intensive care unit were recruited. The global plasma and urine metabolomic profiles were determined by ultr...
2023-04-18 | MTBLS3505 | MetaboLights
UnknownOutbreak of KPC-2-producing Klebsiella pneumoniae endowed with ceftazidime-avibactam resistance mediated through a VEB-1-mutant (VEB-25), Greece, September to October 2019.
Not available
S-EPMC6988274 | biostudies-literature
UnknownCurrent and Potential Therapeutic Options for Infections Caused by Difficult-to-Treat and Pandrug Resistant Gram-Negative Bacteria in Critically Ill Patients.
Not available
S-EPMC9394369 | biostudies-literature
UnknownEvolutionary adaptation revealed by comparative genome analysis of woolly mammoths and elephants.
Not available
S-EPMC5737375 | biostudies-literature
UnknownAge-driven modulation of tRNA-derived fragments in Drosophila and their potential targets.
Not available
S-EPMC4572633 | biostudies-literature
UnknownMultifaceted mechanisms of colistin resistance revealed by genomic analysis of multidrug-resistant Klebsiella pneumoniae isolates from individual patients before and after colistin treatment.
Not available
S-EPMC7264071 | biostudies-literature
UnknownGene replacement therapy in two Golgi-retained CMT1X mutants before and after the onset of demyelinating neuropathy.
Not available
S-EPMC10460951 | biostudies-literature
UnknownThe Use of Genotoxicity Endpoints as Biomarkers of Low Dose Radiation Exposure in Interventional Cardiology.
Not available
S-EPMC8342993 | biostudies-literature
UnknownPopulation pharmacokinetic analysis of colistin methanesulfonate and colistin after intravenous administration in critically ill patients with infections caused by gram-negative bacteria.
Not available
S-EPMC2715599 | biostudies-literature
ProteomicsDefective metabolic programming impairs early neuronal morphogenesis in neural cultures and an organoid model of Leigh syndrome
Leigh syndrome (LS) is a severe manifestation of mitochondrial disease in children and is currently incurable. The lack of effective models hampers our understanding of the mechanisms underlying the neuronal pathology of LS. Using patient-derived induced pluripotent stem cells and CRISPR/Cas9 engine...
ORGANISM(S): Homo sapiens (Human) 
2021-01-29 | PXD019112 | Pride
Mitochondrial disease  leigh syndrome  ipscs  npcs  brain organoids  surf1 
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