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Post-mortem genetic investigation of cardiac disease-associated genes in sudden infant death syndrome (SIDS) cases.
Not available
S-EPMC7782403
|
biostudies-literature
Cite
Variant interpretation in molecular autopsy: a useful dilemma.
Not available
S-EPMC8847204
|
biostudies-literature
Cite
Characterization of an N-terminal Na
v
1.5 channel variant - a potential risk factor for arrhythmias and sudden death?
Not available
S-EPMC7678220
|
biostudies-literature
Cite
Molecular phylogeny and evolution of the cone snails (Gastropoda, Conoidea).
Not available
S-EPMC5556946
|
biostudies-literature
Cite
"Re-evaluation of variants of uncertain significance in patients with hereditary arrhythmogenic disorders".
Not available
S-EPMC11282671
|
biostudies-literature
Cite
Correction: "Re-evaluation of variants of uncertain significance in patients with hereditary arrhythmogenic disorders".
Not available
S-EPMC11529073
|
biostudies-literature
Cite
iNOS expressing macrophages co-localize with nitrotyrosine staining after myocardial infarction in humans.
Not available
S-EPMC10098457
|
biostudies-literature
Cite
Changes in gene expression patterns in postmortem human myocardial infarction.
Not available
S-EPMC7417407
|
biostudies-literature
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The mutation L69P in the PAS domain of the hERG potassium channel results in LQTS by trafficking deficiency.
Not available
S-EPMC7188350
|
biostudies-literature
Cite
Potassium channel TASK-5 forms functional heterodimers with TASK-1 and TASK-3 to break its silence.
Not available
S-EPMC11364637
|
biostudies-literature
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