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ProteomicsProteomics reveals the inhibitory mechanism of Levodopa against esophageal squamous cell carcinoma
The high recurrence rate and poor survival prospects of esophageal squamous cell carcinoma (ESCC) patients after treatment make the ongoing research on chemoprevention drugs for ESCC particularly important. In helping to solve this problem, we screened a large number of FDA-approved drugs and found ...
ORGANISM(S): Homo sapiens (Human) 
2021-09-08 | PXD013824 | Pride
Mitochondria Levodopa Chemoprevention Esophageal squamous cell carcinoma (escc) 
Proteomicsp38 mediated ACSL4 phosphorylation drives stress-induced esophageal squamous cell carcinoma growth through Src myristoylation
Through IP/MS, we discovered that ACSL4 could interact with p38
ORGANISM(S): Homo sapiens (Human) 
2025-03-08 | PXD053908 | Pride
Human escc 
ProteomicsProteomics analysis report of esophageal squamous cell carcinoma treated by Arbidol
In order to detect the effect of Arbidol on the protein expression of esophageal cancer cells, we used control and drug-added KYSE150 cells for proteomic analysis
ORGANISM(S): Homo Sapiens 
2022-06-27 | PXD034944 |
ProteomicsProteomics analysis report of esophageal squamous cell carcinoma treated by Dasabuvir
In order to detect the effect of Dasabuvir on the protein expression of esophageal cancer cells, we used control and drug-added KYSE150 cells for proteomic analysis
ORGANISM(S): Homo Sapiens 
2022-01-24 | PXD031186 |
ProteomicsHMGB2 promote ESCC tumorigenesis and predict neoadjuvant therapy response
Recent studies have demonstrated that chronic stress can enhance the development of multiple human diseases, including cancer. However, the role of chronic stress in esophageal carcinogenesis and its underlying molecular mechanisms remain unclear. This study uncovered that dysregulated cholesterol m...
ORGANISM(S): Mus Musculus 
2025-01-09 | PXD059616 |
ProteomicsChronic stress-induced cholesterol metabolism abnormalities promote ESCC tumorigenesis and predict neoadjuvant therapy response
chronic stress activates the GCR-HMGB2-LDLR axis via cortisol release, leading to abnormal cholesterol metabolism and ultimately promoting esophageal carcinogenesis
ORGANISM(S): Homo Sapiens 
2025-01-09 | PXD059578 |
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