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TranscriptomicsTranscription profiling of mouse normal lymphoid progenitors and MLL-AF4 leukemic lymphoblasts
We created a mouse model where conditional expression of physiologic levels of an Mll-AF4 fusion oncogene induces development of acute lymphoblastic (ALL) or acute myeloid leukemias (AML). Immunophenotypic and gene expression analysis of the ALL cells demonstrated bone marrow replacement with B-prec...
ORGANISM(S): Mus musculus 
2008-11-13 | E-GEOD-12310 | biostudies-arrayexpress
TranscriptomicsTranscription profiling of mouse activated or control 5-FU bone marrow from MLL-AF4stop knockins
We created a mouse model where conditional expression of physiologic levels of an Mll-AF4 fusion oncogene induces development of acute lymphoblastic (ALL) or acute myeloid leukemias (AML). Immunophenotypic and gene expression analysis of the ALL cells demonstrated bone marrow replacement with B-prec...
ORGANISM(S): Mus musculus 
2008-11-13 | E-GEOD-12313 | biostudies-arrayexpress
UnknownEfficacy and safety of biosimilar CT-P17 versus reference adalimumab in subjects with rheumatoid arthritis: 24-week results from a randomized study.
Not available
S-EPMC7863328 | biostudies-literature
UnknownEfficacy and Safety of Biosimilar CT-P47 Versus Reference Tocilizumab: 1-Year Results of a Randomised, Active-Controlled, Double-Blind, Phase III Study in Patients with Rheumatoid Arthritis.
Not available
S-EPMC12307557 | biostudies-literature
UnknownEfficacy and safety of CT-P47 versus reference tocilizumab: 32-week results of a randomised, active-controlled, double-blind, phase III study in patients with rheumatoid arthritis, including 8 weeks of switching data from reference tocilizumab to CT-P47.
Not available
S-EPMC11492937 | biostudies-literature
UnknownEfficacy and safety of switching from reference adalimumab to CT-P17 (100 mg/ml): 52-week randomized, double-blind study in rheumatoid arthritis.
Not available
S-EPMC8996790 | biostudies-literature
TranscriptomicsProfiling transcriptional and translational response to anoxia or hypoxia of genes in DNA damage response and repair pathways in human fibroblast and prostate cancer cells using NanoString nCounter.
Identification of genes in DNA damage response and repair pathways differentially transcribed or translated under anoxia or hypoxia in GM05757 normal human fibroblast cells and DU145 human prostate cancer cells. Comparison of mRNA abundance and translation efficiency of genes in DNA damage response ...
ORGANISM(S): Homo sapiens 
2015-07-01 | E-GEOD-52461 | biostudies-arrayexpress
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