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UnknownmiR526b and miR655 Induce Oxidative Stress in Breast Cancer.
Not available
S-EPMC6720387 | biostudies-literature
UnknownPri-miR526b and Pri-miR655 Are Potential Blood Biomarkers for Breast Cancer.
Not available
S-EPMC8345356 | biostudies-literature
UnknownMir526b and Mir655 Promote Tumour Associated Angiogenesis and Lymphangiogenesis in Breast Cancer.
Not available
S-EPMC6678879 | biostudies-literature
ProteomicsBreast Cancer Cell Secretome Analysis to Decipher miRNA Tumor Biology and Discover Potential Biomarkers
MicroRNA (miRNA/miR) miR526b and miR655 overexpressed tumor cell-free secretions promote breast cancer phenotypes in the tumor microenvironment (TME). However, the mechanisms of miRNA regulating TME have never been investigated. With mass spectrometry analysis of MCF7-miRNA-overexpressed versus miRN...
ORGANISM(S): Homo sapiens (Human) 
2023-05-10 | PXD031771 | Pride
Microrna Bioinformatics Mir655 Mir526b Systems biology Secretome Biomarker Breast cancer Estrogen receptor-positive (er+) Luminal a Tumor microenvironment (tme) 
GenomicsThe Effects of miR-526b, miR655 and COX-2 Overexpression on Oxidative and Genotoxic Stress in MCF7 Breast Cancer Cells
The Effects of miR-526b, miR655 and COX-2 Overexpression on Oxidative and Genotoxic Stress in MCF7 Breast Cancer Cells
PRJNA1254982 | ENA
UnknownChemically Induced Hypoxia Enhances miRNA Functions in Breast Cancer.
Not available
S-EPMC7465874 | biostudies-literature
UnknownBreast cancer cell secretome analysis to decipher miRNA regulating the tumor microenvironment and discover potential biomarkers.
Not available
S-EPMC10148110 | biostudies-literature
TranscriptomicsThe Effects of miR-526b, miR655 and COX-2 Overexpression on Oxidative and Genotoxic Stress in MCF7 Breast Cancer Cells
Breast cancer progression and therapeutic resistance are influenced by the tumor’s ability to adapt to cellular stress, including oxidative damage and chemotherapeutic pressure. Two microRNAs, miR-526b and miR-655, have been shown to promote aggressive breast cancer phenotypes, such as enhanced prol...
ORGANISM(S): Homo sapiens 
2025-09-29 | GSE295502 | GEO
UnknownEP4 as a Therapeutic Target for Aggressive Human Breast Cancer.
Not available
S-EPMC5979567 | biostudies-literature
UnknownDistinct Oxidative Stress Adaptations Driven by the Overexpression of miR-526b, miR-655, and COX-2 in Breast Cancer.
Not available
S-EPMC12470079 | biostudies-literature
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