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ProteomicsRevisiting the relevance of the accumulation of apolipoprotein E (APOE) in mitochondria-associated ER membranes: Investi-gation of hepatic protein-protein interactions of APOE with LONP1, TOMM40 and VDAC1
Investigating the subcellular distribution and protein interaction of APOE and the modulation by cellular stress signals, we aimed to contribute to the elucidation of putative novel functions. We identified APOE as hepatic mitochondria-associated ER-membranes (MAM) protein, where it most likely inte...
ORGANISM(S): Homo sapiens (Human) 
2025-05-07 | PXD055281 | Pride
Mitochondria-er contacts (merc) Mam Stress response Proteome analysis Subcellular distribution Liver Apoe4 
UnknownPerturbed mitochondria-ER contacts in live neurons that model the amyloid pathology of Alzheimer's disease.
Not available
S-EPMC6826005 | biostudies-literature
GenomicsCDK4 Inactivation Hinders Cell Death and Metabolism Remodeling Mitochondria-ER contacts in Triple-Negative Breast cancer cells
CDK4 Inactivation Hinders Cell Death and Metabolism Remodeling Mitochondria-ER contacts in Triple-Negative Breast cancer cells
PRJNA1167725 | ENA
UnknownA Mitofusin-2-dependent inactivating cleavage of Opa1 links changes in mitochondria cristae and ER contacts in the postprandial liver.
Not available
S-EPMC4234614 | biostudies-literature
TranscriptomicsCDK4 Inactivation Hinders Cell Death and Metabolism Remodeling Mitochondria-ER contacts in Triple-Negative Breast cancer cells
The shift in the energetic demands of proliferating cells during tumorigenesis requires extensive crosstalk between the cell cycle and metabolism. Beyond their role in cell proliferation, cell cycle regulators also modulate intracellular metabolism in normal tissues. However, in the context of cance...
ORGANISM(S): Homo sapiens 
2024-10-14 | GSE278524 | GEO
UnknownModulation of Mitochondria-Endoplasmic Reticulum Contacts (MERCs) by Small Molecules as a New Strategy for Restoring Lipid Metabolism in an Amyotrophic Lateral Sclerosis Model.
Not available
S-EPMC11770630 | biostudies-literature
UnknownAltered relaxation and Mitochondria-Endoplasmic Reticulum Contacts Precede Major (Mal)adaptations in Aging Skeletal Muscle and are Prevented by Exercise.
Not available
S-EPMC11838400 | biostudies-literature
UnknownAltered Relaxation and Mitochondria-Endoplasmic Reticulum Contacts Precede Major (Mal)Adaptations in Aging Skeletal Muscle and Are Prevented by Exercise.
Not available
S-EPMC12419855 | biostudies-literature
UnknownDecreasing pdzd8-mediated mito-ER contacts improves organismal fitness and mitigates Aβ<sub>42</sub> toxicity.
Not available
S-EPMC9279675 | biostudies-literature
UnknownRab32 uses its effector reticulon 3L to trigger autophagic degradation of mitochondria-associated membrane (MAM) proteins.
Not available
S-EPMC8573869 | biostudies-literature
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